Role of K(ATP) channels in the induction of ischemic tolerance by the 'adenosine mechanism' in neuronal cultures.
Reshef
A
A
Department of Clinical Biochemistry, Sackler Faculty of Medicine, Tel-Aviv University, Israel.
Sperling
O
O
Zoref-Shani
E
E
D000241:Adenosine / Q000378:metabolism*; D000818:Animals; D002545:Brain Ischemia / Q000378:metabolism*; D002478:Cells, Cultured; D019806:Cromakalim / Q000494:pharmacology; D004075:Diglycerides / Q000494:pharmacology; D005905:Glyburide / Q000494:pharmacology; D009474:Neurons / Q000166:cytology / Q000187:drug effects* / Q000502:physiology; D010660:Phenylisopropyladenosine / Q000494:pharmacology; D026902:Potassium Channel Blockers / Q000494:pharmacology; D015221:Potassium Channels / Q000502:physiology*; D011493:Protein Kinase C / Q000378:metabolism; D051381:Rats
D004075:Diglycerides; D026902:Potassium Channel Blockers; D015221:Potassium Channels; D019806:Cromakalim; C044295:1,2-dioctanoylglycerol; D010660:Phenylisopropyladenosine; D011493:Protein Kinase C; D000241:Adenosine; D005905:Glyburide