Still stressed out but doing fine: normalization of wall stress is superfluous to maintaining cardiac function in chronic pressure overload.
Sano
Motoaki
M
Schneider
Michael D
MD
D000262:Adenylyl Cyclases / Q000378:metabolism; D000818:Animals; D006332:Cardiomegaly / Q000235:genetics / Q000378:metabolism / Q000503:physiopathology*; D003250:Constriction; D017868:Cyclic AMP-Dependent Protein Kinases / Q000378:metabolism; D004299:Dopamine beta-Hydroxylase / Q000235:genetics; D043802:GTP-Binding Protein alpha Subunits, Gq-G11; D006321:Heart / Q000503:physiopathology*; D020962:Heterotrimeric GTP-Binding Proteins / Q000235:genetics; D051379:Mice; D018345:Mice, Knockout; D008822:Mice, Transgenic; D020928:Mitogen-Activated Protein Kinases / Q000378:metabolism; D019869:Phosphatidylinositol 3-Kinases / Q000378:metabolism; D011943:Receptors, Adrenergic, beta / Q000378:metabolism; D015398:Signal Transduction; D051552:beta-Adrenergic Receptor Kinases
D011943:Receptors, Adrenergic, beta; D004299:Dopamine beta-Hydroxylase; D019869:Phosphatidylinositol 3-Kinases; D017868:Cyclic AMP-Dependent Protein Kinases; D051552:beta-Adrenergic Receptor Kinases; D020928:Mitogen-Activated Protein Kinases; D043802:GTP-Binding Protein alpha Subunits, Gq-G11; D020962:Heterotrimeric GTP-Binding Proteins; D000262:Adenylyl Cyclases