Modulation of ventricular arrhythmias by catecholamines during evolving infarction in the isolated perfused rat heart.
Clements-Jewery
H
H
King's Centre for Cardiovascular Biology and Medicine, Rayne Institute, St. Thomas' Hospital, London SE1 7EH, U.K.
Curtis
M J
MJ
D000319:Adrenergic beta-Antagonists / Q000494:pharmacology; D000818:Animals; D001262:Atenolol / Q000494:pharmacology; D002395:Catecholamines / Q000633:toxicity*; D003326:Coronary Circulation / Q000187:drug effects; D004562:Electrocardiography / Q000187:drug effects; D006321:Heart / Q000187:drug effects* / Q000503:physiopathology; D006339:Heart Rate / Q000187:drug effects; D006439:Hemodynamics / Q000187:drug effects / Q000502:physiology; D066298:In Vitro Techniques; D008297:Male; D009121:Muscle Denervation; D009203:Myocardial Infarction / Q000503:physiopathology*; D010879:Piperazines / Q000494:pharmacology; D051381:Rats; D017208:Rats, Wistar; D014665:Vasodilator Agents / Q000494:pharmacology; D014693:Ventricular Fibrillation / Q000139:chemically induced* / Q000503:physiopathology
D000319:Adrenergic beta-Antagonists; D002395:Catecholamines; D010879:Piperazines; D014665:Vasodilator Agents; C005185:trimazosin; D001262:Atenolol