Nitric oxide synthase inhibition decreases output of hepatic insulin sensitizing substance (HISS), which is reversed by SIN-1 but not by nitroprusside.
Guarino
M P
MP
Departamento de Fisiologia-FCM-UNL, Campo Mártires da Pátria, 130, 1169-056 Lisboa, Portugal.
Correia
N C
NC
Raposo
J
J
Macedo
M P
MP
D000818:Animals; D004791:Enzyme Inhibitors / Q000008:administration & dosage / Q000494:pharmacology*; D007275:Injections, Intravenous; D007328:Insulin / Q000097:blood; D007333:Insulin Resistance / Q000502:physiology; D008099:Liver / Q000187:drug effects / Q000378:metabolism*; D008297:Male; D008981:Molsidomine / Q000008:administration & dosage / Q000031:analogs & derivatives / Q000494:pharmacology*; D019331:NG-Nitroarginine Methyl Ester / Q000494:pharmacology; D020030:Nitric Oxide Donors / Q000008:administration & dosage / Q000494:pharmacology*; D019001:Nitric Oxide Synthase / Q000037:antagonists & inhibitors*; D009599:Nitroprusside / Q000008:administration & dosage / Q000494:pharmacology*; D011169:Portal Vein; D011506:Proteins / Q000378:metabolism*; D051381:Rats
D004791:Enzyme Inhibitors; D007328:Insulin; D020030:Nitric Oxide Donors; D011506:Proteins; D009599:Nitroprusside; C002385:linsidomine; D008981:Molsidomine; D019001:Nitric Oxide Synthase; D019331:NG-Nitroarginine Methyl Ester