Cancer research. Leukemia protein spurs gene silencing.
Marx
Jean
J
D001374:Azacitidine / Q000031:analogs & derivatives* / Q000494:pharmacology; D002455:Cell Division; D002471:Cell Transformation, Neoplastic; D004248:DNA (Cytosine-5-)-Methyltransferases / Q000378:metabolism; D019175:DNA Methylation; D020868:Gene Silencing; D016147:Genes, Tumor Suppressor; D006801:Humans; D015473:Leukemia, Promyelocytic, Acute / Q000097:blood / Q000235:genetics* / Q000473:pathology; D007962:Leukocytes / Q000166:cytology; D009154:Mutation; D009363:Neoplasm Proteins / Q000235:genetics / Q000378:metabolism*; D015514:Oncogene Proteins, Fusion / Q000235:genetics / Q000378:metabolism*; D011401:Promoter Regions, Genetic; D018168:Receptors, Retinoic Acid / Q000235:genetics*; D014212:Tretinoin / Q000378:metabolism / Q000494:pharmacology; D015032:Zinc / Q000494:pharmacology
D009363:Neoplasm Proteins; D015514:Oncogene Proteins, Fusion; D018168:Receptors, Retinoic Acid; C093476:promyelocytic leukemia-retinoic acid receptor alpha fusion oncoprotein; C082925:retinoic acid receptor beta; D014212:Tretinoin; C041519:5-aza-2'-deoxycytidine-5'-monophosphate; D004248:DNA (Cytosine-5-)-Methyltransferases; D015032:Zinc; D001374:Azacitidine