Vasomotion and nitric oxide bioactivity in diseased coronary arteries.
Atheromatous coronary stenoses are no longer considered to be passive structures, instead having the capacity for dynamic, often transient, change which may take the form of constriction or dilatation in response to either endogenous or external stimulation.
Tousoulis
D
D
Cardiology Unit, Athens University Medical School, Athens, Greece.
Davies
G J
GJ
Crake
T
T
Toutouzas
P C
PC
D000787:Angina Pectoris / Q000209:etiology; D023921:Coronary Stenosis / Q000378:metabolism* / Q000503:physiopathology; D003329:Coronary Vasospasm / Q000209:etiology; D006801:Humans; D009569:Nitric Oxide / Q000096:biosynthesis*; D014661:Vasoconstriction / Q000502:physiology; D014664:Vasodilation / Q000502:physiology
D009569:Nitric Oxide