Genetic forms of renal potassium and magnesium wasting.
Warnock
David G
DG
D001477:Bartter Syndrome / Q000235:genetics; D002352:Carrier Proteins / Q000235:genetics; D005838:Genotype; D006801:Humans; D007008:Hypokalemia / Q000235:genetics*; D008275:Magnesium Deficiency / Q000235:genetics*; D009154:Mutation; D010641:Phenotype; D015221:Potassium Channels / Q000235:genetics; D024661:Potassium Channels, Inwardly Rectifying; D011955:Receptors, Drug; D015499:Renal Tubular Transport, Inborn Errors / Q000235:genetics*; D049992:Sodium Chloride Symporters; D028021:Sodium-Potassium-Chloride Symporters / Q000235:genetics; D064466:Solute Carrier Family 12, Member 1; D064486:Solute Carrier Family 12, Member 3; D027981:Symporters
D002352:Carrier Proteins; C086989:KCNJ1 protein, human; D015221:Potassium Channels; D024661:Potassium Channels, Inwardly Rectifying; D011955:Receptors, Drug; C578714:SLC12A1 protein, human; C578874:SLC12A3 protein, human; D049992:Sodium Chloride Symporters; D028021:Sodium-Potassium-Chloride Symporters; D064466:Solute Carrier Family 12, Member 1; D064486:Solute Carrier Family 12, Member 3; D027981:Symporters; C064415:thiazide receptor