title: Vasomotion and nitric oxide bioactivity in diseased coronary arteries.
abstract: Atheromatous coronary stenoses are no longer considered to be passive structures, instead having the capacity for dynamic, often transient, change which may take the form of constriction or dilatation in response to either endogenous or external stimulation.
authors.affiliation: Cardiology Unit, Athens University Medical School, Athens, Greece.
authors.forename: D
authors.initials: D
authors.lastname: Tousoulis
authors.forename: G J
authors.initials: GJ
authors.lastname: Davies
authors.forename: T
authors.initials: T
authors.lastname: Crake
authors.forename: P C
authors.initials: PC
authors.lastname: Toutouzas
mesh_terms: D000787:Angina Pectoris / Q000209:etiology; D023921:Coronary Stenosis / Q000378:metabolism* / Q000503:physiopathology; D003329:Coronary Vasospasm / Q000209:etiology; D006801:Humans; D009569:Nitric Oxide / Q000096:biosynthesis*; D014661:Vasoconstriction / Q000502:physiology; D014664:Vasodilation / Q000502:physiology
chemical_list: D009569:Nitric Oxide