Deactivation of vasodilator responses by GRK2 overexpression: a mechanism or the mechanism for hypertension?
Feldman
Ross D
RD
Robarts Research Institute, London, Ontario, Canada. feldmanr@lhsc.on.ca
D017868:Cyclic AMP-Dependent Protein Kinases / Q000187:drug effects / Q000502:physiology*; D019204:GTP-Binding Proteins / Q000502:physiology; D006801:Humans; D006973:Hypertension / Q000188:drug therapy / Q000201:enzymology* / Q000503:physiopathology; D014664:Vasodilation; D014665:Vasodilator Agents / Q000494:pharmacology* / Q000627:therapeutic use; D051552:beta-Adrenergic Receptor Kinases
D014665:Vasodilator Agents; D017868:Cyclic AMP-Dependent Protein Kinases; D051552:beta-Adrenergic Receptor Kinases; D019204:GTP-Binding Proteins