[Processes of coagulation and fibrinolysis in prematurely born neonates with developing respiratory insufficiency].
Neonates deaths occur the most often because of prematurity, dyspnoea and serious infections. Each of the above mentioned complications is accompanied by haemostasis, produced by a destroying effect of a heterologous stimulus on natural anticoagulation mechanism. The aim of the study was to evaluate the activation of the haemostasis system in prematurely born neonates with developing respiratory insufficiency.
Mikulska
Monika
M
Zakład Neonatologii Slaskiej Akademii Medycznej, 41-902 Bytom, ul. Batorego 15. szpitspec2@szpitalspec2.bytom.ids.pl
D000990:Antithrombin III / Q000378:metabolism; D001778:Blood Coagulation Disorders / Q000378:metabolism* / Q000401:mortality / Q000503:physiopathology*; D004797:Enzyme-Linked Immunosorbent Assay; D005338:Fibrin Fibrinogen Degradation Products / Q000378:metabolism; D005342:Fibrinolysis / Q000502:physiology*; D006487:Hemostasis / Q000502:physiology; D006801:Humans; D007231:Infant, Newborn; D007234:Infant, Premature; D010446:Peptide Fragments / Q000378:metabolism; D011486:Protein C / Q000378:metabolism; D011498:Protein Precursors / Q000378:metabolism; D011516:Prothrombin / Q000378:metabolism; D012131:Respiratory Insufficiency / Q000378:metabolism* / Q000401:mortality / Q000503:physiopathology*
D005338:Fibrin Fibrinogen Degradation Products; D010446:Peptide Fragments; D011486:Protein C; D011498:Protein Precursors; C036309:fibrin fragment D; C029733:prothrombin fragment 1; C044183:prothrombin fragment 2; D000990:Antithrombin III; D011516:Prothrombin