Re: Role of endothelial nitric oxide and smooth muscle potassium channels in cerebral arteriolar dilation in response to acidosis.
Rosenblum
William I
WI
D000138:Acidosis / Q000503:physiopathology*; D000818:Animals; D001160:Arterioles / Q000503:physiopathology; D002415:Cats; D002560:Cerebrovascular Circulation / Q000502:physiology; D004305:Dose-Response Relationship, Drug; D004618:Embolism, Air / Q000503:physiopathology; D004730:Endothelium, Vascular / Q000378:metabolism*; D004791:Enzyme Inhibitors / Q000494:pharmacology; D009131:Muscle, Smooth, Vascular / Q000378:metabolism*; D009569:Nitric Oxide / Q000378:metabolism*; D015221:Potassium Channels / Q000378:metabolism*; D051381:Rats; D015203:Reproducibility of Results; D012107:Research Design; D014664:Vasodilation / Q000502:physiology
D004791:Enzyme Inhibitors; D015221:Potassium Channels; D009569:Nitric Oxide