The presence of apoE4, not the absence of apoE3, contributes to AD pathology.
Teter
Bruce
B
UCLA-SFVP MC151 and VA-6LAHS, Sepulveda, CA 91343, USA. bteter@ucla.edu
Raber
Jacob
J
Nathan
Britto
B
Crutcher
Keith A
KA
D000544:Alzheimer Disease / Q000235:genetics / Q000473:pathology*; D000818:Animals; D053318:Apolipoprotein E3; D053327:Apolipoprotein E4; D001057:Apolipoproteins E / Q000235:genetics / Q000378:metabolism*; D001253:Astrocytes / Q000473:pathology; D001921:Brain / Q000473:pathology; D015870:Gene Expression / Q000502:physiology; D006801:Humans; D051379:Mice; D008822:Mice, Transgenic; D009474:Neurons / Q000473:pathology
D053318:Apolipoprotein E3; D053327:Apolipoprotein E4; D001057:Apolipoproteins E