Increased perfusion of the femoral head in transient bone marrow edema syndrome.
The pathophysiology of transient bone marrow edema syndrome is not known. Ischemia has been suggested as the pathophysiologic factor, because the histologic findings are similar to those of early stage osteonecrosis. Angiographic studies of osteonecrotic femoral heads have shown arterial interruption and impaired perfusion. The current report describes the angiographic and scintigraphic findings of transient bone marrow edema syndrome of the hip in a 45-year-old man. The nutrient arteries were dilated, and the femoral head perfusion was increased compared with the unaffected contralateral side. These findings suggest that a vasomotor response plays a role in the pathogenesis of transient bone marrow edema syndrome. The disease might be a reversible process after temporary ischemia of the femoral head.
Koo
Kyung-Hoi
KH
Department of Orthopaedic Surgery, Gyeong-Sang National University School of Medicine, Chinju, South Korea.
Ahn
In-Oak
IO
Song
Hae-Ryong
HR
Kim
Shin-Yoon
SY
Jones
John Paul
JP
D000792:Angiography; D001706:Biopsy; D001855:Bone Marrow Diseases / Q000473:pathology / Q000503:physiopathology*; D004487:Edema / Q000473:pathology / Q000503:physiopathology*; D005270:Femur Head / Q000098:blood supply* / Q000473:pathology; D006801:Humans; D008279:Magnetic Resonance Imaging; D008297:Male; D008875:Middle Aged; D013577:Syndrome