Copper genes are not implicated in the pathogenesis of focal dystonia.
Bandmann
O
O
Department of Neurology, Philipps-University Marburg, Germany. o.bandmann@sheffield.ac.uk
Asmus
F
F
Sibbing
D
D
Grundmann
M
M
Schwab
S G
SG
Müller
J
J
Wildenauer
D B
DB
Poewe
W
W
Gasser
T
T
Oertel
W H
WH
D000251:Adenosine Triphosphatases / Q000235:genetics* / Q000378:metabolism; D000328:Adult; D000368:Aged; D002352:Carrier Proteins / Q000235:genetics* / Q000378:metabolism; D027682:Cation Transport Proteins / Q000235:genetics* / Q000378:metabolism; D003300:Copper / Q000378:metabolism; D000073840:Copper-Transporting ATPases; D020821:Dystonic Disorders / Q000209:etiology / Q000235:genetics*; D005260:Female; D006801:Humans; D008297:Male; D008875:Middle Aged; D009154:Mutation; D011993:Recombinant Fusion Proteins
D002352:Carrier Proteins; D027682:Cation Transport Proteins; D011993:Recombinant Fusion Proteins; D003300:Copper; D000251:Adenosine Triphosphatases; C081184:ATP7A protein, human; D000073840:Copper-Transporting ATPases