Nitric oxide may contribute to the long-term impairment of synaptic transmission after transient ischemia.
D000251:Adenosine Triphosphatases / Q000037:antagonists & inhibitors / Q000378:metabolism; D000818:Animals; D001921:Brain / Q000098:blood supply / Q000503:physiopathology; D002546:Ischemic Attack, Transient / Q000503:physiopathology*; D018377:Neurotransmitter Agents / Q000378:metabolism; D009569:Nitric Oxide / Q000378:metabolism* / Q000494:pharmacology; D020030:Nitric Oxide Donors / Q000494:pharmacology; D030421:Peroxynitrous Acid / Q000378:metabolism / Q000633:toxicity; D010766:Phosphorylation; D051381:Rats; D009435:Synaptic Transmission / Q000187:drug effects; D013995:Time; D005680:gamma-Aminobutyric Acid / Q000378:metabolism
D018377:Neurotransmitter Agents; D020030:Nitric Oxide Donors; D030421:Peroxynitrous Acid; D009569:Nitric Oxide; D005680:gamma-Aminobutyric Acid; D000251:Adenosine Triphosphatases