[Mechanisms by which PPAR gamma modulators regulate insulin sensitivity].
Yamauchi
Toshimasa
T
Department of Metabolic Diseases, Graduate School of Medicine, University of Tokyo.
Kadowaki
Takashi
T
D017667:Adipocytes / Q000166:cytology; D000818:Animals; D017209:Apoptosis; D050882:CREB-Binding Protein; D002454:Cell Differentiation; D003924:Diabetes Mellitus, Type 2 / Q000188:drug therapy / Q000209:etiology / Q000503:physiopathology; D015195:Drug Design; D006801:Humans; D007004:Hypoglycemic Agents; D007333:Insulin Resistance; D009687:Nuclear Proteins / Q000502:physiology; D011110:Polymorphism, Genetic; D018160:Receptors, Cytoplasmic and Nuclear / Q000819:agonists* / Q000037:antagonists & inhibitors / Q000235:genetics / Q000502:physiology; D013844:Thiazoles / Q000494:pharmacology; D015534:Trans-Activators / Q000502:physiology; D014157:Transcription Factors / Q000819:agonists* / Q000037:antagonists & inhibitors / Q000235:genetics / Q000502:physiology
D007004:Hypoglycemic Agents; D009687:Nuclear Proteins; D018160:Receptors, Cytoplasmic and Nuclear; D013844:Thiazoles; D015534:Trans-Activators; D014157:Transcription Factors; D050882:CREB-Binding Protein; C494363:CREBBP protein, human