Overexpression of redox factor-1 protects against postischemic liver injury by reducing oxidative stress and NF-kappa B activity.
Ozaki
M
M
Department of Experimental Surgery and Bioengineering, National Children's Medical Research Center, Tokyo, Japan.
Haga
S
S
Irani
K
K
Amemiya
H
H
Suzuki
S
S
D000256:Adenoviridae / Q000235:genetics; D017209:Apoptosis; D019757:Carbon-Oxygen Lyases / Q000235:genetics*; D002460:Cell Line; D004260:DNA Repair / Q000235:genetics*; D043603:DNA-(Apurinic or Apyrimidinic Site) Lyase; D005822:Genetic Vectors; D006801:Humans; D020672:I-kappa B Proteins / Q000378:metabolism; D007668:Kidney; D008099:Liver / Q000166:cytology / Q000502:physiology; D000072000:NF-KappaB Inhibitor alpha; D016328:NF-kappa B / Q000037:antagonists & inhibitors / Q000378:metabolism*; D018384:Oxidative Stress / Q000502:physiology*; D015427:Reperfusion Injury / Q000523:psychology*; D014162:Transfection
D020672:I-kappa B Proteins; D016328:NF-kappa B; C000606204:NFKBIA protein, human; D000072000:NF-KappaB Inhibitor alpha; D019757:Carbon-Oxygen Lyases; C085108:APEX1 protein, human; D043603:DNA-(Apurinic or Apyrimidinic Site) Lyase