Use of nucleotide excision repair-deficient mice as a model for chemically induced lung cancer.
Cheo
David L
DL
Life Technologies Division, Invitrogen Corporation, Rockville, MD, USA.
Friedberg
Errol C
EC
D015073:2-Acetylaminofluorene / Q000633:toxicity*; D000818:Animals; D002273:Carcinogens / Q000633:toxicity*; D003043:Cocarcinogenesis; D003433:Crosses, Genetic; D017931:DNA Primers / Q000737:chemistry; D004260:DNA Repair; D004268:DNA-Binding Proteins / Q000172:deficiency / Q000235:genetics*; D004195:Disease Models, Animal; D005260:Female; D020022:Genetic Predisposition to Disease; D005838:Genotype; D006579:Heterozygote; D006720:Homozygote; D006801:Humans; D006881:Hydroxyacetylaminofluorene / Q000633:toxicity; D008175:Lung Neoplasms / Q000139:chemically induced / Q000209:etiology* / Q000235:genetics*; D008297:Male; D051379:Mice; D018345:Mice, Knockout; D016133:Polymerase Chain Reaction; D016159:Tumor Suppressor Protein p53 / Q000172:deficiency / Q000235:genetics*
D002273:Carcinogens; D017931:DNA Primers; D004268:DNA-Binding Proteins; D016159:Tumor Suppressor Protein p53; C501002:Xpc protein, mouse; C086984:XPC protein, human; D006881:Hydroxyacetylaminofluorene; D015073:2-Acetylaminofluorene