[Lipotoxicity as a new mechanism in metabolic syndrome. Does this explain insulin resistance?].
Müller-Wieland
D
D
Deutsches Diabetes Forschungsinstitut, Leibniz-Institut, Heinrich-Heine-Universität Düsseldorf. mueller-wieland@ddfi.uniduesseldorf.de
Kotzka
J
J
D000273:Adipose Tissue / Q000503:physiopathology*; D000818:Animals; D022762:CCAAT-Enhancer-Binding Proteins / Q000502:physiology; D004268:DNA-Binding Proteins / Q000502:physiology; D003920:Diabetes Mellitus / Q000503:physiopathology*; D006801:Humans; D007333:Insulin Resistance / Q000502:physiology*; D050356:Lipid Metabolism; D008055:Lipids / Q000633:toxicity*; D024821:Metabolic Syndrome / Q000503:physiopathology*; D051379:Mice; D009765:Obesity; D015398:Signal Transduction / Q000502:physiology; D051780:Sterol Regulatory Element Binding Protein 1; D014157:Transcription Factors; D015431:Weight Loss / Q000502:physiology
D022762:CCAAT-Enhancer-Binding Proteins; D004268:DNA-Binding Proteins; D008055:Lipids; C497387:SREBF1 protein, human; C497388:Srebf1 protein, mouse; D051780:Sterol Regulatory Element Binding Protein 1; D014157:Transcription Factors