Stress signals activate natural killer cells.
Long
Eric O
EO
Laboratory of Immunogenetics, National Institute of Allergy and Infectious Diseases, National Institutes of Health, 12441 Parklawn Drive, Rockville, MD 20852, USA. elong@nih.gov
Rajagopalan
Sumati
S
D015703:Antigens, CD / Q000502:physiology*; D018834:Chaperonin 60 / Q000378:metabolism*; D006680:HLA Antigens / Q000378:metabolism*; D015395:Histocompatibility Antigens Class I / Q000378:metabolism*; D006801:Humans; D007694:Killer Cells, Natural / Q000276:immunology*; D037181:Lectins, C-Type / Q000502:physiology*; D008213:Lymphocyte Activation; D051920:NK Cell Lectin-Like Receptor Subfamily D; D021382:Protein Sorting Signals / Q000502:physiology*; D011971:Receptors, Immunologic / Q000502:physiology*; D054340:Receptors, KIR; D055607:Receptors, Natural Killer Cell; D000097625:HLA-E Antigens
D015703:Antigens, CD; D018834:Chaperonin 60; D006680:HLA Antigens; D015395:Histocompatibility Antigens Class I; D037181:Lectins, C-Type; D051920:NK Cell Lectin-Like Receptor Subfamily D; D021382:Protein Sorting Signals; D011971:Receptors, Immunologic; D054340:Receptors, KIR; D055607:Receptors, Natural Killer Cell; D000097625:HLA-E Antigens