The molecular mechanisms of allergic diseases: immunoglobulin E dependent and independent signaling pathways converge in eliciting the release of arachidonic acid metabolites.
Sagi-Eisenberg
Ronit
R
Department of Cell Biology and Histology, Sackler Faculty of Medicine, Tel Aviv University, Ramat Aviv, Israel. histol3@post.tau.ac.il
D016718:Arachidonic Acid / Q000276:immunology* / Q000378:metabolism*; D020962:Heterotrimeric GTP-Binding Proteins / Q000276:immunology; D006801:Humans; D006967:Hypersensitivity / Q000276:immunology* / Q000378:metabolism*; D007073:Immunoglobulin E / Q000276:immunology* / Q000378:metabolism*; D007249:Inflammation; D018836:Inflammation Mediators / Q000276:immunology; D008407:Mast Cells / Q000276:immunology / Q000378:metabolism; D011505:Protein-Tyrosine Kinases / Q000276:immunology; D017455:Receptors, IgE / Q000276:immunology; D015398:Signal Transduction / Q000276:immunology*
D018836:Inflammation Mediators; D017455:Receptors, IgE; D016718:Arachidonic Acid; D007073:Immunoglobulin E; D011505:Protein-Tyrosine Kinases; D020962:Heterotrimeric GTP-Binding Proteins