title: Are PU.1 mutations frequent genetic events in acute myeloid leukemia (AML)?
authors.forename: Charlotte
authors.initials: C
authors.lastname: Lamandin
authors.forename: Christophe
authors.initials: C
authors.lastname: Sagot
authors.forename: Christophe
authors.initials: C
authors.lastname: Roumier
authors.forename: Pascale
authors.initials: P
authors.lastname: Lepelley
authors.forename: Stéphane
authors.initials: S
authors.lastname: De Botton
authors.forename: Alain
authors.initials: A
authors.lastname: Cosson
authors.forename: Pierre
authors.initials: P
authors.lastname: Fenaux
authors.forename: Claude
authors.initials: C
authors.lastname: Preudhomme
mesh_terms: D000208:Acute Disease; D004252:DNA Mutational Analysis; D018076:DNA, Complementary / Q000235:genetics; D004273:DNA, Neoplasm / Q000235:genetics; D005006:Ethnicity / Q000235:genetics; D006801:Humans; D007951:Leukemia, Myeloid / Q000235:genetics*; D009363:Neoplasm Proteins / Q000235:genetics*; D018807:Polymorphism, Single-Stranded Conformational; D011518:Proto-Oncogene Proteins / Q000235:genetics*; D017422:Sequence Analysis, DNA; D017384:Sequence Deletion; D015534:Trans-Activators / Q000235:genetics*; D000099232:Proto-Oncogene Protein Spi-1
chemical_list: D018076:DNA, Complementary; D004273:DNA, Neoplasm; D009363:Neoplasm Proteins; D011518:Proto-Oncogene Proteins; D015534:Trans-Activators; D000099232:Proto-Oncogene Protein Spi-1