Hepatocytes transgenic for the full hepatitis C virus open reading frame are resistant to fas-induced cell death.
Disson
Olivier
O
Institut de Génétique Moléculaire, CNRS UMR 5535, F-34293 Montpellier Cedex 5, France.
Hahne
Michael
M
Hibner
Urszula
U
Lerat
Herve
H
D000818:Animals; D000970:Antineoplastic Agents / Q000494:pharmacology; D016923:Cell Death / Q000187:drug effects / Q000502:physiology*; D002478:Cells, Cultured; D016174:Hepacivirus / Q000235:genetics*; D022781:Hepatocytes / Q000166:cytology* / Q000187:drug effects; D007191:Indazoles / Q000494:pharmacology; D051379:Mice; D008822:Mice, Transgenic; D008928:Mitochondria / Q000502:physiology; D016366:Open Reading Frames; D011994:Recombinant Proteins / Q000494:pharmacology; D019014:fas Receptor / Q000494:pharmacology / Q000502:physiology*
D000970:Antineoplastic Agents; D007191:Indazoles; D011994:Recombinant Proteins; D019014:fas Receptor; C016371:lonidamine