Triamcinolone acetonide and dexamethasome suppress TNF-alpha-induced histone H4 acetylation on lysine residues 8 and 12 in mononuclear cells.
Tsaprouni
Loukia G
LG
Department of Biology Health Science, University of Luton, Luton LU1 3JU, UK. ltsapr@hotmail.com
Ito
Kazuhiro
K
Punchard
Neville
N
Adcock
Ian M
IM
D000107:Acetylation; D000893:Anti-Inflammatory Agents / Q000494:pharmacology*; D003907:Dexamethasone / Q000494:pharmacology*; D056572:Histone Deacetylase Inhibitors; D006655:Histone Deacetylases / Q000235:genetics; D006657:Histones / Q000378:metabolism*; D006801:Humans; D016209:Interleukin-8 / Q000502:physiology; D008239:Lysine; D009000:Monocytes / Q000166:cytology / Q000187:drug effects / Q000378:metabolism*; D014222:Triamcinolone Acetonide / Q000494:pharmacology*; D014409:Tumor Necrosis Factor-alpha / Q000494:pharmacology* / Q000502:physiology; D020298:U937 Cells
D000893:Anti-Inflammatory Agents; D056572:Histone Deacetylase Inhibitors; D006657:Histones; D016209:Interleukin-8; D014409:Tumor Necrosis Factor-alpha; D003907:Dexamethasone; D006655:Histone Deacetylases; D014222:Triamcinolone Acetonide; D008239:Lysine