Overexpression of Na+/Ca2+ exchanger attenuates postinfarcted myocardial dysfunction.
Min
Jiang-Yong
JY
Cardiovascular Division, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts 02146, USA.
Sullivan
Matthew F
MF
Chu
Victor
V
Wang
Ju-Feng
JF
Amende
Ivo
I
Morgan
James P
JP
Philipson
Kenneth D
KD
Hampton
Thomas G
TG
D000318:Adrenergic beta-Agonists / Q000494:pharmacology; D000818:Animals; D004195:Disease Models, Animal; D005786:Gene Expression Regulation / Q000187:drug effects; D006439:Hemodynamics / Q000502:physiology; D007545:Isoproterenol / Q000494:pharmacology; D051379:Mice; D008822:Mice, Transgenic; D009203:Myocardial Infarction / Q000503:physiopathology* / Q000517:prevention & control; D019831:Sodium-Calcium Exchanger / Q000235:genetics*
D000318:Adrenergic beta-Agonists; D019831:Sodium-Calcium Exchanger; D007545:Isoproterenol