title: Overexpression of Na+/Ca2+ exchanger attenuates postinfarcted myocardial dysfunction.
authors.affiliation: Cardiovascular Division, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts 02146, USA.
authors.forename: Jiang-Yong
authors.initials: JY
authors.lastname: Min
authors.forename: Matthew F
authors.initials: MF
authors.lastname: Sullivan
authors.forename: Victor
authors.initials: V
authors.lastname: Chu
authors.forename: Ju-Feng
authors.initials: JF
authors.lastname: Wang
authors.forename: Ivo
authors.initials: I
authors.lastname: Amende
authors.forename: James P
authors.initials: JP
authors.lastname: Morgan
authors.forename: Kenneth D
authors.initials: KD
authors.lastname: Philipson
authors.forename: Thomas G
authors.initials: TG
authors.lastname: Hampton
mesh_terms: D000318:Adrenergic beta-Agonists / Q000494:pharmacology; D000818:Animals; D004195:Disease Models, Animal; D005786:Gene Expression Regulation / Q000187:drug effects; D006439:Hemodynamics / Q000502:physiology; D007545:Isoproterenol / Q000494:pharmacology; D051379:Mice; D008822:Mice, Transgenic; D009203:Myocardial Infarction / Q000503:physiopathology* / Q000517:prevention & control; D019831:Sodium-Calcium Exchanger / Q000235:genetics*
chemical_list: D000318:Adrenergic beta-Agonists; D019831:Sodium-Calcium Exchanger; D007545:Isoproterenol