The thiazide-sensitive na-cl cotransporter and human disease: reemergence of an old player.
Ellison
David H
DH
Division of Nephrology & Hypertension, Oregon Health & Science University, and VA Medical Center, Portland, Oregon.
D000471:Alkalosis / Q000235:genetics / Q000503:physiopathology*; D000818:Animals; D000959:Antihypertensive Agents / Q000627:therapeutic use; D001581:Benzothiadiazines; D001794:Blood Pressure / Q000187:drug effects / Q000235:genetics; D002352:Carrier Proteins / Q000037:antagonists & inhibitors / Q000235:genetics / Q000378:metabolism*; D004232:Diuretics; D006801:Humans; D006973:Hypertension / Q000188:drug therapy; D007008:Hypokalemia / Q000235:genetics / Q000503:physiopathology*; D009154:Mutation; D011955:Receptors, Drug / Q000037:antagonists & inhibitors / Q000235:genetics / Q000378:metabolism*; D049993:Sodium Chloride Symporter Inhibitors / Q000627:therapeutic use; D049992:Sodium Chloride Symporters; D064486:Solute Carrier Family 12, Member 3; D027981:Symporters; D013577:Syndrome
D000959:Antihypertensive Agents; D001581:Benzothiadiazines; D002352:Carrier Proteins; D004232:Diuretics; D011955:Receptors, Drug; C578874:SLC12A3 protein, human; D049993:Sodium Chloride Symporter Inhibitors; D049992:Sodium Chloride Symporters; D064486:Solute Carrier Family 12, Member 3; D027981:Symporters; C064415:thiazide receptor