A patient with sodium- and potassium-losing nephropathy.
A young man with salt-losing nephropathy had the unusual coexistence of hypokalemia caused by secondary hyperaldosteronism. When his NaCl intake was supplemented by 12 g/day (205 mmol), hyperaldosteronism was suppressed and so were his urinary K wasting, hypokalemia, and episodic falling, during an extended follow-up. His findings are compared and contrasted with those in other reported patients with renal salt wasting, and also those in patients with Gitelman and Bartter syndromes.
Fulop
Milford
M
Department of Medicine, Albert Einstein College of Medicine and Jacobi Medical Center, Bronx, New York, USA. fulop@aecom.yu.edu
D000293:Adolescent; D000328:Adult; D000368:Aged; D001774:Blood Chemical Analysis; D019587:Dietary Supplements; D005260:Female; D006801:Humans; D007008:Hypokalemia / Q000378:metabolism; D007010:Hyponatremia / Q000378:metabolism; D007674:Kidney Diseases / Q000473:pathology* / Q000503:physiopathology*; D008297:Male; D008875:Middle Aged; D011188:Potassium / Q000378:metabolism*; D012964:Sodium / Q000378:metabolism*
D012964:Sodium; D011188:Potassium