Coagulopathies and arterial stroke.
Although hypercoagulable states are most often associated with venous thrombosis, arterial thromboses are reported in protein S, protein C, and antithrombin III deficiencies, factor V Leiden and prothrombin gene mutations, hyperhomocysteinemia, dysfibrinogenemia, plasminogen deficiency, sickle cell disease, and antiphospholipid antibody syndrome.
Moster
Mark L
ML
Department of Neurosensory Sciences, Albert Einstein Medical Center, Philadelphia, USA. mmoster@aol.com
D000755:Anemia, Sickle Cell / Q000209:etiology; D016736:Antiphospholipid Syndrome / Q000276:immunology / Q000503:physiopathology; D001796:Blood Protein Disorders / Q000145:classification / Q000150:complications / Q000235:genetics / Q000378:metabolism; D004211:Disseminated Intravascular Coagulation / Q000150:complications*; D005165:Factor V / Q000235:genetics / Q000378:metabolism; D015241:Fibrinogens, Abnormal / Q000235:genetics / Q000378:metabolism; D006801:Humans; D020138:Hyperhomocysteinemia / Q000209:etiology / Q000235:genetics; D010958:Plasminogen / Q000235:genetics / Q000378:metabolism; D011516:Prothrombin / Q000235:genetics / Q000378:metabolism; D020521:Stroke / Q000209:etiology / Q000235:genetics / Q000378:metabolism*
D015241:Fibrinogens, Abnormal; C095381:factor V Leiden; D005165:Factor V; D011516:Prothrombin; D010958:Plasminogen