[Animal models for abnormal purine metabolism].
Morisaki
Takayuki
T
Department of Bioscience, National Cardiovascular Center Research Institute, Department of Molecular Pathophysiology, Osaka Unversity Graduate School of Pharmaceutical Sciences.
D000659:AMP Deaminase / Q000172:deficiency; D000228:Adenine Phosphoribosyltransferase / Q000172:deficiency; D000243:Adenosine Deaminase / Q000172:deficiency; D000818:Animals; D004195:Disease Models, Animal; D006073:Gout / Q000209:etiology; D006801:Humans; D033461:Hyperuricemia / Q000209:etiology; D007041:Hypoxanthine Phosphoribosyltransferase / Q000172:deficiency; D007926:Lesch-Nyhan Syndrome / Q000209:etiology; D051379:Mice; D018345:Mice, Knockout; D011683:Purine-Nucleoside Phosphorylase / Q000172:deficiency; D011686:Purine-Pyrimidine Metabolism, Inborn Errors; D011687:Purines / Q000378:metabolism; D014503:Urate Oxidase / Q000172:deficiency; D014968:Xanthine Dehydrogenase / Q000172:deficiency
D011687:Purines; D014968:Xanthine Dehydrogenase; D014503:Urate Oxidase; D011683:Purine-Nucleoside Phosphorylase; D000228:Adenine Phosphoribosyltransferase; D007041:Hypoxanthine Phosphoribosyltransferase; D000243:Adenosine Deaminase; D000659:AMP Deaminase; C030985:purine