title: Genetic heterogeneity of autoimmune disorders in the nonobese diabetic mouse.
abstract: The nonobese diabetic mouse is highly susceptible not only to diabetes but to several autoimmune diseases, and one might suspect that these are controlled by a shared set of genes. However, based on various gene-segregation experiments, it seems that only a few loci are shared and that each disorder is influenced also by a unique set of genes.
authors.affiliation: Section for Medical Inflammation Research, Department of Cell and Molecular Biology, University of Lund, Sweden. asa.johansson@inflam.lu.se
authors.forename: A C M
authors.initials: AC
authors.lastname: Johansson
authors.forename: A-K B
authors.initials: AK
authors.lastname: Lindqvist
authors.forename: M
authors.initials: M
authors.lastname: Johannesson
authors.forename: R
authors.initials: R
authors.lastname: Holmdahl
mesh_terms: D000818:Animals; D001327:Autoimmune Diseases / Q000235:genetics* / Q000276:immunology; D005260:Female; D020022:Genetic Predisposition to Disease; D014644:Genetic Variation / Q000276:immunology; D008285:Major Histocompatibility Complex / Q000235:genetics / Q000276:immunology; D008297:Male; D051379:Mice; D016688:Mice, Inbred NOD / Q000235:genetics* / Q000276:immunology; D040641:Quantitative Trait Loci / Q000276:immunology