Effects of the G protein beta 3-subunit gene C825T polymorphism: should hypotheses regarding the molecular mechanisms underlying enhanced G protein activation be revised? Focus on "A splice variant of the G protein beta 3-subunit implicated in disease states does not modulate ion channels".
Siffert
Winfried
W
D017398:Alternative Splicing / Q000235:genetics*; D000818:Animals; D003596:Cytosine / Q000378:metabolism; D014644:Genetic Variation / Q000235:genetics*; D020962:Heterotrimeric GTP-Binding Proteins / Q000235:genetics* / Q000378:metabolism; D006801:Humans; D007473:Ion Channels / Q000235:genetics* / Q000378:metabolism; D011110:Polymorphism, Genetic / Q000235:genetics*; D021122:Protein Subunits / Q000235:genetics* / Q000378:metabolism; D013941:Thymine / Q000378:metabolism; D014652:Vascular Diseases / Q000235:genetics
C454420:G-protein beta3 subunit; D007473:Ion Channels; D021122:Protein Subunits; D003596:Cytosine; D020962:Heterotrimeric GTP-Binding Proteins; D013941:Thymine