Inflammation and vascular hypertrophy induced by angiotensin II: role of NADPH oxidase-derived reactive oxygen species independently of blood pressure elevation?
Ernesto L
EL
Schiffrin
Rhian M
RM
Touyz
D000804:Angiotensin II / Q000008:administration & dosage / Q000502:physiology / Q000633:toxicity*; D000818:Animals; D004730:Endothelium, Vascular / Q000378:metabolism* / Q000473:pathology; D004789:Enzyme Activation; D004791:Enzyme Inhibitors / Q000008:administration & dosage / Q000633:toxicity*; D005786:Gene Expression Regulation; D006023:Glycoproteins / Q000008:administration & dosage / Q000633:toxicity*; D006801:Humans; D006984:Hypertrophy; D008562:Membrane Glycoproteins / Q000037:antagonists & inhibitors* / Q000502:physiology; D000074662:NADPH Oxidase 2; D019255:NADPH Oxidases; D021122:Protein Subunits; D051381:Rats; D017382:Reactive Oxygen Species / Q000378:metabolism*; D013481:Superoxides / Q000378:metabolism; D014657:Vasculitis / Q000378:metabolism*
D004791:Enzyme Inhibitors; D006023:Glycoproteins; D008562:Membrane Glycoproteins; D021122:Protein Subunits; D017382:Reactive Oxygen Species; C473031:gp91ds-tat protein, chimeric; D013481:Superoxides; D000804:Angiotensin II; C056558:CYBB protein, human; D000074662:NADPH Oxidase 2; D019255:NADPH Oxidases