title: How important is GAD in the etiology of spontaneous disease in human and murine type 1 diabetes?
authors.affiliation: Torrey Pines Institute for Molecular Studies, 3350 General Atomics Court, San Diego, CA 92121-1122, USA.
authors.forename: Gerald
authors.initials: G
authors.lastname: Nepom
authors.forename: Anthony
authors.initials: A
authors.lastname: Quinn
authors.forename: Eli
authors.initials: E
authors.lastname: Sercarz
authors.forename: Darcy B
authors.initials: DB
authors.lastname: Wilson
mesh_terms: D000818:Animals; D001324:Autoantigens; D003922:Diabetes Mellitus, Type 1 / Q000201:enzymology* / Q000209:etiology* / Q000276:immunology; D005968:Glutamate Decarboxylase / Q000276:immunology*; D006801:Humans; D008213:Lymphocyte Activation; D051379:Mice; D016688:Mice, Inbred NOD; D011236:Prediabetic State / Q000201:enzymology / Q000209:etiology / Q000276:immunology; D013601:T-Lymphocytes / Q000276:immunology
chemical_list: D001324:Autoantigens; D005968:Glutamate Decarboxylase