Post-traumatic inflammation following spinal cord injury.
Inflammatory reaction following a spinal cord injury (SCI) contributes substantially to secondary effects, with both beneficial and devastating effects. This review summarizes the current knowledge concerning the structural features (vascular, cellular, and biochemical events) of SCI and gives an overview of the regulation of post-traumatic inflammation.
Hausmann
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ON
Neurosurgical Department, University Clinics Basel, Switzerland.
D000818:Animals; D002118:Calcium / Q000378:metabolism; D016923:Cell Death; D005609:Free Radicals / Q000009:adverse effects; D005904:Glial Fibrillary Acidic Protein / Q000378:metabolism; D018698:Glutamic Acid / Q000009:adverse effects; D006801:Humans; D007249:Inflammation / Q000209:etiology / Q000378:metabolism; D008928:Mitochondria / Q000378:metabolism; D009457:Neuroglia / Q000378:metabolism*; D009474:Neurons / Q000378:metabolism*; D009498:Neurotoxins / Q000009:adverse effects; D009569:Nitric Oxide / Q000378:metabolism; D013119:Spinal Cord Injuries / Q000378:metabolism* / Q000473:pathology / Q000503:physiopathology; D014855:Wallerian Degeneration / Q000378:metabolism
D005609:Free Radicals; D005904:Glial Fibrillary Acidic Protein; D009498:Neurotoxins; D009569:Nitric Oxide; D018698:Glutamic Acid; D002118:Calcium