The Th1 and Th2 concept in atopic allergic disease.
Robinson
D S
DS
Imperial College School of Medicine, National Heart and Lung Institute, London, UK. d.s.robinson@ic.ac.uk
D000485:Allergens / Q000009:adverse effects; D000818:Animals; D001249:Asthma / Q000276:immunology; D002454:Cell Differentiation; D018925:Chemokines / Q000494:pharmacology / Q000502:physiology; D016207:Cytokines / Q000235:genetics / Q000502:physiology; D004195:Disease Models, Animal; D006801:Humans; D006969:Hypersensitivity, Immediate / Q000276:immunology*; D007073:Immunoglobulin E / Q000096:biosynthesis; D007378:Interleukins / Q000502:physiology; D051379:Mice; D008807:Mice, Inbred BALB C; D018345:Mice, Knockout; D012333:RNA, Messenger / Q000096:biosynthesis; D019707:Receptors, Chemokine / Q000187:drug effects / Q000502:physiology; D018417:Th1 Cells / Q000276:immunology*; D018418:Th2 Cells / Q000276:immunology*; D014157:Transcription Factors / Q000502:physiology; D014158:Transcription, Genetic
D000485:Allergens; D018925:Chemokines; D016207:Cytokines; D007378:Interleukins; D012333:RNA, Messenger; D019707:Receptors, Chemokine; D014157:Transcription Factors; D007073:Immunoglobulin E