Long-lived C. elegans daf-2 mutants are resistant to bacterial pathogens.
Garsin
Danielle A
DA
Department of Genetics, Massachusetts General Hospital, Boston, MA 02114, USA.
Villanueva
Jacinto M
JM
Begun
Jakob
J
Kim
Dennis H
DH
Sifri
Costi D
CD
Calderwood
Stephen B
SB
Ruvkun
Gary
G
Ausubel
Frederick M
FM
D000483:Alleles; D000818:Animals; D017173:Caenorhabditis elegans / Q000235:genetics / Q000276:immunology / Q000382:microbiology* / Q000502:physiology*; D029742:Caenorhabditis elegans Proteins / Q000235:genetics / Q000502:physiology; D013293:Enterococcus faecalis / Q000472:pathogenicity* / Q000502:physiology; D004926:Escherichia coli / Q000472:pathogenicity* / Q000502:physiology; D051858:Forkhead Transcription Factors; D008136:Longevity; D009154:Mutation; D010641:Phenotype; D019869:Phosphatidylinositol 3-Kinases / Q000235:genetics / Q000502:physiology; D011972:Receptor, Insulin / Q000235:genetics / Q000502:physiology*; D013211:Staphylococcus aureus / Q000472:pathogenicity* / Q000502:physiology; D014157:Transcription Factors / Q000235:genetics / Q000502:physiology
D029742:Caenorhabditis elegans Proteins; D051858:Forkhead Transcription Factors; D019869:Phosphatidylinositol 3-Kinases; D011972:Receptor, Insulin; D014157:Transcription Factors; C108831:daf-16 protein, C elegans; C100662:AGE-1 protein, C elegans; C107483:DAF-2 protein, C elegans