Aspirin resistance is not a common biochemical phenotype explained by unblocked cyclooxygenase-1 activity.
Hillarp
A
A
Lethagen
S
S
Mattiasson
I
I
D000255:Adenosine Triphosphate / Q000096:biosynthesis / Q000097:blood; D000328:Adult; D001241:Aspirin / Q000494:pharmacology*; D051545:Cyclooxygenase 1; D016861:Cyclooxygenase Inhibitors / Q000494:pharmacology; D004351:Drug Resistance; D005260:Female; D006801:Humans; D007527:Isoenzymes / Q000037:antagonists & inhibitors / Q000378:metabolism*; D008565:Membrane Proteins; D008875:Middle Aged; D010641:Phenotype; D010974:Platelet Aggregation / Q000187:drug effects / Q000502:physiology; D011451:Prostaglandin-Endoperoxide Synthases / Q000378:metabolism*
D016861:Cyclooxygenase Inhibitors; D007527:Isoenzymes; D008565:Membrane Proteins; D000255:Adenosine Triphosphate; D051545:Cyclooxygenase 1; C496511:PTGS1 protein, human; D011451:Prostaglandin-Endoperoxide Synthases; D001241:Aspirin