[Angio-MR assessment of vasomotor reactivity impairment as a long-term outcome of SAH following intracerebral aneurysm rupture].
Studies on long-term outcome of subarachnoid hemorrhage (SAH) have been carried out for many years using various neuroimaging techniques, such as e.g.: SPECT, PET, TCD and XeCT. In our study angio-MRI supplemented with the acetazolamide test was used to assess cerebrovascular reserve impairment in 30 patients within 6 months since clipping an intracranial aneurysm. Severity of the SAH course was evaluated using the WFNS (World Federation of Neurosurgical Societies) scale [3]. The patients' clinical status was assessed at follow-up by means of the Glasgow Outcome Scale (GOS) [8]. Cereberovascular reserve evaluated at the follow-up in hypercapneal conditions was found to be insufficient. The degree of vessel reactivity dysfunction as a long-term outcome of SAH turned out to depend on massiveness of hemorrhage from the ruptured aneurysm.
Smida
Karina
K
Katedry i Kliniki Neurochirurgii Akademii Medycznej w Gdańsku.
Słoniewski
Paweł
P
D000086:Acetazolamide; D017542:Aneurysm, Ruptured / Q000150:complications* / Q000473:pathology* / Q000503:physiopathology; D000927:Anticonvulsants; D001783:Blood Flow Velocity / Q000502:physiology; D005500:Follow-Up Studies; D015600:Glasgow Coma Scale; D006801:Humans; D006935:Hypercapnia / Q000175:diagnosis / Q000209:etiology; D002532:Intracranial Aneurysm / Q000150:complications* / Q000473:pathology* / Q000503:physiopathology; D018810:Magnetic Resonance Angiography; D009131:Muscle, Smooth, Vascular / Q000473:pathology* / Q000503:physiopathology; D013345:Subarachnoid Hemorrhage / Q000209:etiology* / Q000473:pathology* / Q000503:physiopathology
D000927:Anticonvulsants; D000086:Acetazolamide