[Antroduodenal motility in patients with gastroesophageal reflux disease].
Beside the defective lower esophageal sphincter different mechanisms may be involved pathophysiology of GERD. This study shows antroduodenal motility disorders including a lower incidence of IMC's and lower frequencies of contractions in some study periods as possible pathophysiologic feature in selected patients with atypical symptoms and increased duodenogastric reflux.
D000328:Adult; D000368:Aged; D004383:Duodenogastric Reflux / Q000503:physiopathology; D004386:Duodenum / Q000503:physiopathology; D005260:Female; D005745:Gastric Acidity Determination; D005764:Gastroesophageal Reflux / Q000503:physiopathology*; D005769:Gastrointestinal Motility / Q000502:physiology*; D006801:Humans; D008297:Male; D008365:Manometry; D008875:Middle Aged; D018670:Monitoring, Ambulatory; D011706:Pyloric Antrum / Q000503:physiopathology; D012815:Signal Processing, Computer-Assisted