[Tissue protection by ischemic preconditioning depends mainly on expression of heat shock protein 32].
Using the hairless mouse ear skin flap model we demonstrate that HSP-32 expression has to be considered as a significant mechanism of ischemic preconditioning-induced protection of critically perfused tissue.
D000818:Animals; D001783:Blood Flow Velocity / Q000502:physiology; D006085:Graft Survival / Q000502:physiology*; D006419:Heme Oxygenase (Decyclizing) / Q000502:physiology*; D051547:Heme Oxygenase-1; D019194:Ischemic Preconditioning / Q000379:methods*; D017078:Laser-Doppler Flowmetry; D008565:Membrane Proteins; D051379:Mice; D008812:Mice, Hairless; D008833:Microcirculation / Q000503:physiopathology; D008866:Microsurgery; D013524:Surgical Flaps / Q000098:blood supply*
D008565:Membrane Proteins; D006419:Heme Oxygenase (Decyclizing); D051547:Heme Oxygenase-1; C496542:Hmox1 protein, mouse