title: Heat-exposed pig islets are protected from inflammatory mediators but express proapoptotic proteins.
authors.affiliation: Third Medical Department, University Hospital, Giessen, Germany.
authors.forename: D
authors.initials: D
authors.lastname: Brandhorst
authors.forename: H
authors.initials: H
authors.lastname: Brandhorst
authors.forename: V
authors.initials: V
authors.lastname: Kumarasamy
authors.forename: A
authors.initials: A
authors.lastname: Maataoui
authors.forename: M D
authors.initials: MD
authors.lastname: Brendel
authors.forename: R G
authors.initials: RG
authors.lastname: Bretzel
mesh_terms: D000818:Animals; D002469:Cell Separation / Q000379:methods; D002470:Cell Survival; D006358:Hot Temperature; D006979:Hyperthermia, Induced; D007249:Inflammation; D007328:Insulin / Q000378:metabolism; D000078790:Insulin Secretion; D007515:Islets of Langerhans / Q000166:cytology / Q000378:metabolism; D023421:Models, Animal; D013552:Swine; D014021:Tissue Preservation / Q000379:methods
chemical_list: D007328:Insulin