Assessing the role of multiple phosphoinositide 3-kinases in chemokine signaling: use of dominant negative mutants controlled by a tetracycline-regulated gene expression system.
Department of Pharmacy and Pharmacology, University of Bath, Bath, U.K.
Adam P
AP
Curnock
Yannis
Y
Sotsios
Stephen G
SG
Ward
D001483:Base Sequence; D018925:Chemokines / Q000378:metabolism*; D015870:Gene Expression; D005821:Genetic Techniques; D006801:Humans; D007527:Isoenzymes / Q000235:genetics / Q000378:metabolism; D019169:Jurkat Cells; D009154:Mutation; D019869:Phosphatidylinositol 3-Kinases / Q000235:genetics* / Q000378:metabolism*; D010957:Plasmids / Q000235:genetics; D012097:Repressor Proteins / Q000235:genetics; D015398:Signal Transduction; D013753:Tetracycline Resistance / Q000235:genetics; D014162:Transfection
D018925:Chemokines; D007527:Isoenzymes; D012097:Repressor Proteins; C034811:tetracycline resistance-encoding transposon repressor protein; D019869:Phosphatidylinositol 3-Kinases