title: Jaundice in low birthweight infants: pathobiology and outcome.
abstract: Jaundice in preterm, as well as full term, infants results from (a) an increased bilirubin load in the hepatocyte, (b) decreased hepatic uptake of bilirubin from the plasma, and/or (c) defective bilirubin conjugation. Hyperbilirubinaemia in preterm infants is more prevalent, more severe, and its course more protracted than in term neonates.
authors: Division of Neonatology and Developmental Biology, Department of Pediatrics, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania 15213, USA. jwatchko@mail.magee.edu
authors: J F
authors: JF
authors: Watchko
authors: M J
authors: MJ
authors: Maisels
mesh_terms: D001663:Bilirubin / Q000378:metabolism; D006801:Humans; D006932:Hyperbilirubinemia / Q000150:complications / Q000378:metabolism / Q000628:therapy; D007230:Infant, Low Birth Weight; D007231:Infant, Newborn; D007235:Infant, Premature, Diseases / Q000209:etiology* / Q000378:metabolism / Q000628:therapy; D007567:Jaundice, Neonatal / Q000209:etiology* / Q000378:metabolism / Q000628:therapy; D007647:Kernicterus / Q000209:etiology; D010789:Phototherapy / Q000379:methods; D011379:Prognosis; D012307:Risk Factors
chemical_list: D001663:Bilirubin