title: Reperfusion injury of pancreas allografts: relation to islet cell function.
abstract: It is unknown to what extent preservation and/or reperfusion may damage islet cells in pancreas allografts. In this study, the release of insulin after reperfusion was used as a marker of injury to the islet cell and compared with the best insulin secretory response (ISR) after glucagon stimulation over a period of 100 days after pancreas transplantation.
authors: Department of Nephrology and Transplantation, University Hospital Maastricht, Maastricht, The Netherlands.
authors: H G
authors: HG
authors: Peltenburg
authors: B H
authors: BH
authors: Wolffenbuttel
authors: M H
authors: MH
authors: Booster
authors: P P
authors: PP
authors: Menheere
authors: K M
authors: KM
authors: Leunissen
authors: G
authors: G
authors: Kootstra
authors: J P
authors: JP
authors: van Hooff
mesh_terms: D000328:Adult; D005260:Female; D005934:Glucagon / Q000494:pharmacology; D006084:Graft Rejection / Q000503:physiopathology*; D006801:Humans; D007328:Insulin / Q000097:blood / Q000378:metabolism*; D000078790:Insulin Secretion; D007515:Islets of Langerhans / Q000378:metabolism*; D016030:Kidney Transplantation / Q000473:pathology*; D008297:Male; D008875:Middle Aged; D010179:Pancreas / Q000098:blood supply; D016035:Pancreas Transplantation / Q000473:pathology*; D015427:Reperfusion Injury
chemical_list: D007328:Insulin; D005934:Glucagon