How to activate a damage-tolerant polymerase: consequences of PCNA modifications by ubiquitin and SUMO.
Ulrich
Helle D
HD
Max Planck Institute for Terrestrial Microbiology, Karl-von-Frisch-Strasse, Marburg, Germany. hulrich@staff.uni-marburg.de
D050759:Cyclin-Dependent Kinase Inhibitor p21; D016213:Cyclins / Q000378:metabolism; D004260:DNA Repair / Q000235:genetics / Q000502:physiology*; D004259:DNA-Directed DNA Polymerase / Q000235:genetics / Q000378:metabolism*; D004789:Enzyme Activation; D008961:Models, Structural; D018809:Proliferating Cell Nuclear Antigen / Q000235:genetics / Q000378:metabolism*; D012568:Schizosaccharomyces / Q000235:genetics / Q000378:metabolism; D025841:Small Ubiquitin-Related Modifier Proteins / Q000378:metabolism; D025801:Ubiquitin / Q000235:genetics / Q000378:metabolism*
D050759:Cyclin-Dependent Kinase Inhibitor p21; D016213:Cyclins; D018809:Proliferating Cell Nuclear Antigen; D025841:Small Ubiquitin-Related Modifier Proteins; D025801:Ubiquitin; D004259:DNA-Directed DNA Polymerase