[Lymphoproliferative disorders caused by hereditary genetic defects].
Kanegane
Hirokazu
H
Department of Pediatrics, Faculty of Medicine, Toyama Medical and Pharmaceutical University.
Miyawaki
Toshio
T
D000818:Animals; D017209:Apoptosis; D001327:Autoimmune Diseases; D002352:Carrier Proteins / Q000235:genetics*; D053222:Fas Ligand Protein; D030342:Genetic Diseases, Inborn / Q000150:complications*; D008040:Genetic Linkage; D006801:Humans; D047908:Intracellular Signaling Peptides and Proteins; D008232:Lymphoproliferative Disorders / Q000209:etiology* / Q000235:genetics* / Q000276:immunology; D008562:Membrane Glycoproteins / Q000235:genetics; D051379:Mice; D000071179:Signaling Lymphocytic Activation Molecule Associated Protein; D014960:X Chromosome
D002352:Carrier Proteins; C505661:FASLG protein, human; D053222:Fas Ligand Protein; C505658:Fasl protein, mouse; D047908:Intracellular Signaling Peptides and Proteins; D008562:Membrane Glycoproteins; C114953:SH2D1A protein, human; C499569:Sh2d1a protein, mouse; D000071179:Signaling Lymphocytic Activation Molecule Associated Protein