Increased particulate partitioning of PKC epsilon reverses susceptibility of phospholamban knockout hearts to ischemic injury.
D000818:Animals; D002135:Calcium-Binding Proteins / Q000172:deficiency / Q000235:genetics / Q000502:physiology*; D003433:Crosses, Genetic; D020022:Genetic Predisposition to Disease / Q000235:genetics*; D051379:Mice; D008815:Mice, Inbred Strains; D018345:Mice, Knockout; D017202:Myocardial Ischemia / Q000235:genetics / Q000503:physiopathology* / Q000517:prevention & control*; D011493:Protein Kinase C / Q000302:isolation & purification / Q000378:metabolism*; D051744:Protein Kinase C-epsilon; D000099211:Phospholamban
D002135:Calcium-Binding Proteins; D011493:Protein Kinase C; D051744:Protein Kinase C-epsilon; D000099211:Phospholamban; C497256:Prkce protein, mouse