Conditional ablation of the neural cell adhesion molecule reduces precision of spatial learning, long-term potentiation, and depression in the CA1 subfield of mouse hippocampus.
D000818:Animals; D054732:Calcium-Calmodulin-Dependent Protein Kinase Type 2; D017871:Calcium-Calmodulin-Dependent Protein Kinases / Q000235:genetics; D004558:Electric Stimulation; D004594:Electrophysiology; D018390:Gene Targeting; D006624:Hippocampus / Q000378:metabolism / Q000502:physiology*; D007859:Learning Disabilities / Q000235:genetics*; D017774:Long-Term Potentiation / Q000235:genetics*; D036881:Long-Term Synaptic Depression / Q000235:genetics*; D018782:Maze Learning / Q000502:physiology*; D008568:Memory; D051379:Mice; D018345:Mice, Knockout; D008817:Mice, Mutant Strains; D008822:Mice, Transgenic; D019006:Neural Cell Adhesion Molecules / Q000172:deficiency / Q000235:genetics*; D009473:Neuronal Plasticity / Q000235:genetics; D011401:Promoter Regions, Genetic / Q000235:genetics
D019006:Neural Cell Adhesion Molecules; D054732:Calcium-Calmodulin-Dependent Protein Kinase Type 2; D017871:Calcium-Calmodulin-Dependent Protein Kinases