Frontonasal process-specific disruption of AP-2alpha results in postnatal midfacial hypoplasia, vascular anomalies, and nasal cavity defects.
D000818:Animals; D001483:Base Sequence; D001808:Blood Vessels / Q000002:abnormalities*; D017931:DNA Primers; D004268:DNA-Binding Proteins / Q000235:genetics / Q000502:physiology*; D005145:Face / Q000002:abnormalities* / Q000098:blood supply; D006801:Humans; D018878:Limb Buds / Q000254:growth & development; D051379:Mice; D009296:Nasal Cavity / Q000002:abnormalities*; D050656:Transcription Factor AP-2; D014157:Transcription Factors / Q000235:genetics / Q000502:physiology*
D017931:DNA Primers; D004268:DNA-Binding Proteins; C493673:TFAP2A protein, human; C493668:Tfap2a protein, mouse; D050656:Transcription Factor AP-2; D014157:Transcription Factors