Activation of the aryl hydrocarbon receptor diminishes the memory response to homotypic influenza virus infection but does not impair host resistance.
D000281:Administration, Intranasal; D000818:Animals; D000914:Antibodies, Viral / Q000096:biosynthesis*; D001992:Bronchoalveolar Lavage Fluid / Q000276:immunology; D018414:CD8-Positive T-Lymphocytes / Q000187:drug effects* / Q000276:immunology; D004785:Environmental Pollutants / Q000633:toxicity*; D005260:Female; D016867:Immunocompromised Host; D007156:Immunologic Memory / Q000187:drug effects*; D016130:Immunophenotyping; D009980:Influenza A virus / Q000276:immunology* / Q000472:pathogenicity; D008198:Lymph Nodes / Q000276:immunology; D051379:Mice; D008810:Mice, Inbred C57BL; D000072317:Polychlorinated Dibenzodioxins / Q000633:toxicity*; D018336:Receptors, Aryl Hydrocarbon / Q000276:immunology* / Q000378:metabolism; D013997:Time Factors
D000914:Antibodies, Viral; D004785:Environmental Pollutants; D000072317:Polychlorinated Dibenzodioxins; D018336:Receptors, Aryl Hydrocarbon