Translational induction of the inhibitor of apoptosis protein HIAP2 during endoplasmic reticulum stress attenuates cell death and is mediated via an inducible internal ribosome entry site element.
D017209:Apoptosis; D015153:Blotting, Western; D020169:Caspases / Q000378:metabolism; D016923:Cell Death; D002460:Cell Line; D002470:Cell Survival; D015500:Chloramphenicol O-Acetyltransferase / Q000378:metabolism; D015536:Down-Regulation; D004721:Endoplasmic Reticulum / Q000378:metabolism*; D017353:Gene Deletion; D006367:HeLa Cells; D006801:Humans; D015151:Immunoblotting; D051034:Inhibitor of Apoptosis Proteins; D016366:Open Reading Frames; D010957:Plasmids / Q000378:metabolism; D011233:Precipitin Tests; D014176:Protein Biosynthesis; D011506:Proteins; D012260:Ribonucleases / Q000378:metabolism; D012270:Ribosomes / Q000378:metabolism*; D014158:Transcription, Genetic; D014162:Transfection; D014415:Tunicamycin / Q000494:pharmacology; D044767:Ubiquitin-Protein Ligases; D014466:Ultraviolet Rays; D015854:Up-Regulation; D001616:beta-Galactosidase / Q000378:metabolism
D051034:Inhibitor of Apoptosis Proteins; D011506:Proteins; D014415:Tunicamycin; D015500:Chloramphenicol O-Acetyltransferase; C494833:BIRC2 protein, human; D044767:Ubiquitin-Protein Ligases; D012260:Ribonucleases; D001616:beta-Galactosidase; D020169:Caspases