Enhanced neuronal excitability in the absence of neurodegeneration induces cerebellar ataxia.
D000818:Animals; D001030:Apamin / Q000378:metabolism; D002524:Cerebellar Ataxia / Q000473:pathology / Q000503:physiopathology*; D002529:Cerebellar Nuclei / Q000166:cytology / Q000473:pathology / Q000503:physiopathology*; D004594:Electrophysiology; D066298:In Vitro Techniques; D051379:Mice; D008822:Mice, Transgenic; D009043:Motor Activity; D009474:Neurons / Q000473:pathology* / Q000502:physiology*; D015221:Potassium Channels / Q000235:genetics / Q000378:metabolism; D011401:Promoter Regions, Genetic; D011689:Purkinje Cells / Q000166:cytology / Q000502:physiology; D011993:Recombinant Fusion Proteins / Q000235:genetics / Q000378:metabolism; D019076:Transgenes
D015221:Potassium Channels; D011993:Recombinant Fusion Proteins; D001030:Apamin