The protective role of intracellular GSH status in the arsenite-induced vascular endothelial dysfunction.
D000111:Acetylcysteine / Q000494:pharmacology; D000818:Animals; D017209:Apoptosis; D018053:Arsenites / Q000633:toxicity*; D002478:Cells, Cultured; D004730:Endothelium, Vascular / Q000166:cytology / Q000187:drug effects* / Q000502:physiology; D005434:Flow Cytometry; D005978:Glutathione / Q000378:metabolism*; D048031:JNK Mitogen-Activated Protein Kinases; D048670:MAP Kinase Kinase 4; D020929:Mitogen-Activated Protein Kinase Kinases / Q000378:metabolism; D015398:Signal Transduction; D013552:Swine; D018808:Transcription Factor AP-1 / Q000378:metabolism; D015854:Up-Regulation
D018053:Arsenites; D018808:Transcription Factor AP-1; D048031:JNK Mitogen-Activated Protein Kinases; D048670:MAP Kinase Kinase 4; D020929:Mitogen-Activated Protein Kinase Kinases; D005978:Glutathione; C015001:arsenite; D000111:Acetylcysteine