Deletion of the alternatively spliced fibronectin EIIIA domain in mice reduces atherosclerosis.
D017398:Alternative Splicing / Q000235:genetics; D000818:Animals; D000831:Animals, Newborn; D001011:Aorta / Q000473:pathology / Q000648:ultrastructure; D001057:Apolipoproteins E / Q000172:deficiency / Q000235:genetics; D001161:Arteriosclerosis / Q000235:genetics* / Q000378:metabolism* / Q000517:prevention & control; D002784:Cholesterol / Q000097:blood; D004041:Dietary Fats; D005091:Exons / Q000235:genetics; D005109:Extracellular Matrix / Q000378:metabolism; D005260:Female; D005347:Fibroblasts / Q000378:metabolism; D005353:Fibronectins / Q000097:blood / Q000172:deficiency / Q000235:genetics / Q000502:physiology*; D005487:Foam Cells / Q000378:metabolism / Q000473:pathology / Q000648:ultrastructure; D017353:Gene Deletion; D006801:Humans; D008079:Lipoproteins, VLDL / Q000378:metabolism; D008297:Male; D051379:Mice; D008807:Mice, Inbred BALB C; D008810:Mice, Inbred C57BL; D018345:Mice, Knockout; D020033:Protein Isoforms; D017434:Protein Structure, Tertiary
D001057:Apolipoproteins E; D004041:Dietary Fats; D005353:Fibronectins; D008079:Lipoproteins, VLDL; D020033:Protein Isoforms; D002784:Cholesterol