A membrane transport defect leads to a rapid attenuation of translation initiation in Saccharomyces cerevisiae.
D002478:Cells, Cultured; D002746:Chlorpromazine / Q000494:pharmacology; D007425:Intracellular Membranes / Q000378:metabolism*; D008565:Membrane Proteins / Q000378:metabolism; D009154:Mutation / Q000235:genetics; D010448:Peptide Initiation Factors / Q000378:metabolism; D014176:Protein Biosynthesis / Q000187:drug effects / Q000235:genetics*; D011493:Protein Kinase C / Q000378:metabolism; D011494:Protein Kinases / Q000378:metabolism; D017346:Protein Serine-Threonine Kinases; D021381:Protein Transport / Q000502:physiology; D012343:RNA, Transfer / Q000096:biosynthesis; D012270:Ribosomes / Q000378:metabolism*; D012441:Saccharomyces cerevisiae / Q000235:genetics / Q000378:metabolism*; D029701:Saccharomyces cerevisiae Proteins; D022161:Transport Vesicles / Q000378:metabolism*
D008565:Membrane Proteins; D010448:Peptide Initiation Factors; C119383:SLG1 protein, S cerevisiae; D029701:Saccharomyces cerevisiae Proteins; C410795:eIF4E-associated protein Eap1; D012343:RNA, Transfer; D011494:Protein Kinases; C512912:GCN2 protein, S cerevisiae; D017346:Protein Serine-Threonine Kinases; D011493:Protein Kinase C; D002746:Chlorpromazine