Premature stop codons involved in muscular dystrophies show a broad spectrum of readthrough efficiencies in response to gentamicin treatment.
D016475:3T3 Cells; D000818:Animals; D000900:Anti-Bacterial Agents / Q000627:therapeutic use*; D018389:Codon, Nonsense; D018388:Codon, Terminator; D003131:Combined Modality Therapy; D018274:Electroporation; D015870:Gene Expression; D015316:Genetic Therapy / Q000379:methods*; D005839:Gentamicins / Q000627:therapeutic use*; D008156:Luciferases / Q000235:genetics; D008297:Male; D051379:Mice; D008810:Mice, Inbred C57BL; D018482:Muscle, Skeletal / Q000201:enzymology*; D009136:Muscular Dystrophies / Q000188:drug therapy / Q000628:therapy*; D001616:beta-Galactosidase / Q000235:genetics
D000900:Anti-Bacterial Agents; D018389:Codon, Nonsense; D018388:Codon, Terminator; D005839:Gentamicins; D008156:Luciferases; D001616:beta-Galactosidase