Counteracting the Nogo receptor enhances optic nerve regeneration if retinal ganglion cells are in an active growth state.
D000818:Animals; D002470:Cell Survival / Q000235:genetics; D046508:Culture Techniques; D000229:Dependovirus / Q000235:genetics; D005260:Female; D058851:GPI-Linked Proteins; D005799:Genes, Dominant; D017930:Genes, Reporter; D015316:Genetic Therapy / Q000379:methods; D005822:Genetic Vectors / Q000235:genetics; D049452:Green Fluorescent Proteins; D008164:Luminescent Proteins / Q000096:biosynthesis / Q000235:genetics; D009185:Myelin Proteins; D009409:Nerve Crush; D009416:Nerve Regeneration / Q000187:drug effects* / Q000502:physiology; D000070816:Nogo Receptor 1; D009900:Optic Nerve / Q000187:drug effects* / Q000254:growth & development; D020221:Optic Nerve Injuries / Q000188:drug therapy* / Q000503:physiopathology; D051381:Rats; D017207:Rats, Sprague-Dawley; D011956:Receptors, Cell Surface; D018000:Receptors, Peptide / Q000037:antagonists & inhibitors* / Q000235:genetics / Q000502:physiology; D012165:Retinal Ganglion Cells / Q000187:drug effects* / Q000502:physiology*; D013997:Time Factors; D014162:Transfection
D058851:GPI-Linked Proteins; D008164:Luminescent Proteins; D009185:Myelin Proteins; D000070816:Nogo Receptor 1; D011956:Receptors, Cell Surface; D018000:Receptors, Peptide; C486581:Rtn4r protein, rat; D049452:Green Fluorescent Proteins