Hypertensive myocardial fibrosis and diastolic dysfunction: another model of inflammation?
D000818:Animals; D000911:Antibodies, Monoclonal / Q000494:pharmacology / Q000627:therapeutic use; D001024:Aortic Valve Stenosis / Q000150:complications / Q000235:genetics; D018932:Chemokine CCL2 / Q000037:antagonists & inhibitors / Q000096:biosynthesis / Q000235:genetics / Q000276:immunology; D003971:Diastole; D004719:Endomyocardial Fibrosis / Q000209:etiology / Q000503:physiopathology*; D005347:Fibroblasts / Q000502:physiology*; D006973:Hypertension / Q000150:complications*; D006984:Hypertrophy; D017379:Hypertrophy, Left Ventricular / Q000209:etiology / Q000235:genetics / Q000473:pathology; D007249:Inflammation; D008264:Macrophages / Q000502:physiology*; D008297:Male; D008955:Models, Cardiovascular; D032383:Myocytes, Cardiac / Q000473:pathology; D012333:RNA, Messenger / Q000096:biosynthesis / Q000235:genetics; D051381:Rats; D017208:Rats, Wistar; D016212:Transforming Growth Factor beta / Q000096:biosynthesis / Q000235:genetics
D000911:Antibodies, Monoclonal; D018932:Chemokine CCL2; D012333:RNA, Messenger; D016212:Transforming Growth Factor beta