Possible role of apoptosis in the pathogenesis of bleomycin-induced scleroderma.
D000818:Animals; D000903:Antibiotics, Antineoplastic; D017209:Apoptosis; D001761:Bleomycin; D053148:Caspase 3; D020169:Caspases / Q000235:genetics / Q000378:metabolism; D020405:Dermis / Q000378:metabolism / Q000473:pathology; D004195:Disease Models, Animal; D053222:Fas Ligand Protein; D005260:Female; D015870:Gene Expression; D020287:In Situ Nick-End Labeling; D008562:Membrane Glycoproteins / Q000235:genetics / Q000378:metabolism; D051379:Mice; D008809:Mice, Inbred C3H; D012595:Scleroderma, Systemic / Q000139:chemically induced / Q000209:etiology* / Q000473:pathology*; D012598:Sclerosis; D013047:Specific Pathogen-Free Organisms; D019014:fas Receptor / Q000235:genetics / Q000378:metabolism
D000903:Antibiotics, Antineoplastic; D053222:Fas Ligand Protein; C505658:Fasl protein, mouse; D008562:Membrane Glycoproteins; D019014:fas Receptor; D001761:Bleomycin; C505409:Casp3 protein, mouse; D053148:Caspase 3; D020169:Caspases