[Baboon syndrome].
Andersen et al described baboon syndrome in 1984. It was characterized by a clinical presentation of systemic contact dermatitis with pruritic and confluent maculopapular light-red eruption, localized in the gluteal area and the major flexures, developed several hours or days after drug or agent contact. This syndrome has a pathognomonic distribution but its cause has not been elucidated yet. Histopathology of the lesions shows non-specific features of dermatitis. Several drugs have been previously described as responsible for the Baboon syndrome origin. Mercury is the most frequent implicated agent; other agents are nickel, different antibiotics, heparine, aminophylline, pseudoephedrine, terbinafine and immunoglobulins
Servicio de Alergia, Clínica Puerta de Hierro, Madrid, Spain. lsanchezmorillas@hotmail.com
L
L
Sánchez-Morillas
M
M
Reaño Martos
M
M
Rodríguez Mosquera
C
C
Iglesias Cadarso
L
L
González Sánchez
A R
AR
Domínguez Lázaro
D000293:Adolescent; D000328:Adult; D000368:Aged; D002081:Buttocks; D017449:Dermatitis, Allergic Contact / Q000145:classification* / Q000209:etiology / Q000473:pathology; D004342:Drug Hypersensitivity / Q000145:classification* / Q000209:etiology / Q000473:pathology; D004890:Erythema / Q000209:etiology / Q000473:pathology; D005260:Female; D006801:Humans; D008297:Male; D008875:Middle Aged; D010328:Patch Tests; D011537:Pruritus / Q000209:etiology / Q000473:pathology; D013577:Syndrome