The effects of an autocrine loop mediated by platelet-activating factor (PAF) in HEC-1A cells are reverted by uteroglobin.
Department of Clinical Physiopathology, Endocrine Unit, University of Florence, Italy.
Alessandro
A
Peri
Lorella
L
Bonaccorsi
Federica
F
Cioppi
Monica
M
Muratori
Michaela
M
Luconi
Simone
S
Granchi
Mario
M
Maggi
Elisabetta
E
Baldi
D000230:Adenocarcinoma / Q000378:metabolism* / Q000473:pathology; D000818:Animals; D019898:Autocrine Communication / Q000502:physiology*; D002455:Cell Division; D045744:Cell Line, Tumor; D016889:Endometrial Neoplasms / Q000378:metabolism* / Q000473:pathology; D005260:Female; D006801:Humans; D020928:Mitogen-Activated Protein Kinases / Q000378:metabolism; D010741:Phospholipases A / Q000037:antagonists & inhibitors; D010972:Platelet Activating Factor / Q000096:biosynthesis / Q000502:physiology*; D011493:Protein Kinase C / Q000378:metabolism; D014598:Uteroglobin / Q000502:physiology*
D010972:Platelet Activating Factor; D014598:Uteroglobin; D011493:Protein Kinase C; D020928:Mitogen-Activated Protein Kinases; D010741:Phospholipases A