[Pathological study on the mechanism of plaque and thrombus formations in the coronary artery of patients with acute coronary syndrome].
Ehara
Shoichi
S
Yoshikawa
Junichi
J
Ueda
Makiko
M
D000789:Angina, Unstable / Q000209:etiology* / Q000473:pathology*; D001161:Arteriosclerosis / Q000378:metabolism / Q000473:pathology; D002455:Cell Division; D002465:Cell Movement; D003328:Coronary Thrombosis / Q000209:etiology* / Q000473:pathology; D003331:Coronary Vessels / Q000166:cytology / Q000378:metabolism / Q000473:pathology*; D016757:Death, Sudden, Cardiac / Q000209:etiology* / Q000473:pathology*; D004730:Endothelium, Vascular / Q000166:cytology / Q000378:metabolism / Q000473:pathology; D005109:Extracellular Matrix / Q000378:metabolism / Q000473:pathology; D005487:Foam Cells / Q000473:pathology; D006801:Humans; D008077:Lipoproteins, LDL / Q000378:metabolism; D009131:Muscle, Smooth, Vascular / Q000166:cytology / Q000378:metabolism / Q000473:pathology; D009203:Myocardial Infarction / Q000209:etiology* / Q000473:pathology*; D010974:Platelet Aggregation; D019039:Platelet Glycoprotein GPIIb-IIIa Complex / Q000502:physiology; D013577:Syndrome
D008077:Lipoproteins, LDL; D019039:Platelet Glycoprotein GPIIb-IIIa Complex; C099252:oxidized low density lipoprotein