Over-expression of rexA nullifies T4rII exclusion in Escherichia coli K(lambda) lysogens.
Dosage and relative cellular levels of RexA and RexB proteins encoded by the rexA-rexB genes of a lambda prophage are important for the Rex+ phenotype, which was nullified when greater RexA or RexB was provided than was necessary for the complementation of a rexA- or a rexB- prophage.
Slavcev
Roderick A
RA
Department of Microbiology and Immunology, University of Saskatchewan, Saskatoon, Saskatchewan S7N 5E5, Canada.
Hayes
Sidney
S
D017122:Bacteriophage T4 / Q000235:genetics* / Q000254:growth & development; D010582:Bacteriophage lambda / Q000235:genetics* / Q000502:physiology; D004926:Escherichia coli / Q000821:virology*; D017353:Gene Deletion; D015870:Gene Expression; D005814:Genes, Viral / Q000235:genetics / Q000502:physiology; D005816:Genetic Complementation Test; D008242:Lysogeny; D009154:Mutation; D010957:Plasmids; D039002:Prophages / Q000235:genetics / Q000502:physiology; D017361:Viral Nonstructural Proteins / Q000235:genetics*; D014764:Viral Proteins / Q000235:genetics*
C094107:RexA protein, Bacteriophage lambda; D017361:Viral Nonstructural Proteins; D014764:Viral Proteins; C065296:rIIA protein, Enterobacteria phage T4; C069331:rexB protein, Bacteriophage lambda