Apoptosis and plaque destabilization in atherosclerosis: the role of macrophage apoptosis induced by cholesterol.
Tabas
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Department of Medicine, Columbia University, New York, NY 10032, USA. iat1@columbia.edu
D000818:Animals; D017209:Apoptosis / Q000502:physiology*; D001157:Arterial Occlusive Diseases / Q000209:etiology; D001161:Arteriosclerosis / Q000150:complications / Q000378:metabolism / Q000503:physiopathology*; D020169:Caspases / Q000378:metabolism; D002478:Cells, Cultured; D002784:Cholesterol / Q000378:metabolism / Q000502:physiology*; D002788:Cholesterol Esters / Q000378:metabolism; D008078:Cholesterol, LDL / Q000378:metabolism; D004721:Endoplasmic Reticulum / Q000378:metabolism / Q000502:physiology; D004730:Endothelium, Vascular / Q000378:metabolism / Q000473:pathology / Q000503:physiopathology; D006801:Humans; D007653:Ketocholesterols / Q000378:metabolism / Q000502:physiology; D008077:Lipoproteins, LDL / Q000378:metabolism / Q000502:physiology; D008264:Macrophages / Q000378:metabolism*; D008954:Models, Biological; D017510:Protein Folding; D015398:Signal Transduction / Q000502:physiology; D002785:Sterol O-Acyltransferase / Q000378:metabolism / Q000502:physiology
D002788:Cholesterol Esters; D008078:Cholesterol, LDL; D007653:Ketocholesterols; D008077:Lipoproteins, LDL; C099252:oxidized low density lipoprotein; D002784:Cholesterol; D002785:Sterol O-Acyltransferase; D020169:Caspases