Calponin (CaP) as a latch-bridge protein--a new concept in regulation of contractility in smooth muscles.
Szymanski
Pawel T
PT
Physiology Program, Department of Environmental Health, Harvard School of Public Health, 665 Huntington Avenue, Boston, MA 02115, USA. pszymans@hsph.harvard.edu
D000199:Actins / Q000502:physiology; D000818:Animals; D017301:Ca(2+) Mg(2+)-ATPase / Q000037:antagonists & inhibitors / Q000378:metabolism; D002135:Calcium-Binding Proteins / Q000494:pharmacology / Q000502:physiology*; D006801:Humans; D066298:In Vitro Techniques; D008840:Microfilament Proteins; D009119:Muscle Contraction / Q000187:drug effects / Q000502:physiology*; D009130:Muscle, Smooth / Q000187:drug effects / Q000502:physiology*; D009218:Myosins / Q000378:metabolism; D010766:Phosphorylation; D013997:Time Factors; D000096985:Calponins
D000199:Actins; D017301:Ca(2+) Mg(2+)-ATPase; D002135:Calcium-Binding Proteins; D008840:Microfilament Proteins; D009218:Myosins; D000096985:Calponins