Tumor-induced dysfunction in T lymphocytes: increased sensitivity to apoptosis.
Finke
J H
JH
Department of Immunology, Lerner Research Institute, The Cleveland Clinic Foundation, Cleveland, OH 44195, USA. finkej@ccf.org
Tannenbaum
C
C
Storkus
W
W
Rayman
P
P
Das
T
T
Biswas
K
K
Richmond
A
A
Moon
C
C
Thornton
M
M
Gill
I
I
Novick
A
A
Bukowski
R
R
D000447:Aldehydes / Q000378:metabolism; D017209:Apoptosis / Q000276:immunology*; D002292:Carcinoma, Renal Cell / Q000276:immunology*; D002851:Chromatography, High Pressure Liquid; D002855:Chromatography, Thin Layer; D005732:Gangliosides / Q000378:metabolism; D006801:Humans; D020287:In Situ Nick-End Labeling; D066298:In Vitro Techniques; D007680:Kidney Neoplasms / Q000276:immunology*; D018384:Oxidative Stress / Q000276:immunology; D017382:Reactive Oxygen Species / Q000378:metabolism; D013601:T-Lymphocytes / Q000276:immunology*; D019139:Tumor Escape / Q000276:immunology
D000447:Aldehydes; D005732:Gangliosides; D017382:Reactive Oxygen Species; C027576:4-hydroxy-2-nonenal