Melatonin enhances retinoic acid induction of cone arrestin gene expression in retinoblastoma cells.
Li
Aimin
A
The Mary D. Allen Laboratory for Vision Research, Doheny Eye Institute, Department of Cell and Neurobiology, Keck School of Medicine of the University of Southern California, BMT401, 1333 San Pablo Street, Los Angeles, CA 90089-9112, USA.
Zhu
Xuemei
X
Brown
Bruce
B
Craft
Cheryl M
CM
D019393:Arrestin / Q000235:genetics / Q000378:metabolism*; D045744:Cell Line, Tumor; D004305:Dose-Response Relationship, Drug; D004357:Drug Synergism; D005786:Gene Expression Regulation / Q000187:drug effects; D006801:Humans; D008550:Melatonin / Q000008:administration & dosage / Q000494:pharmacology*; D057094:Nuclear Receptor Subfamily 1, Group F, Member 1; D011401:Promoter Regions, Genetic / Q000187:drug effects / Q000502:physiology; D012333:RNA, Messenger / Q000378:metabolism; D018160:Receptors, Cytoplasmic and Nuclear / Q000235:genetics; D017949:Retinal Cone Photoreceptor Cells / Q000378:metabolism*; D019572:Retinal Neoplasms / Q000235:genetics / Q000378:metabolism*; D012175:Retinoblastoma / Q000235:genetics / Q000378:metabolism*; D013997:Time Factors; D015534:Trans-Activators / Q000235:genetics; D014212:Tretinoin / Q000008:administration & dosage / Q000494:pharmacology*
D019393:Arrestin; D057094:Nuclear Receptor Subfamily 1, Group F, Member 1; D012333:RNA, Messenger; D018160:Receptors, Cytoplasmic and Nuclear; D015534:Trans-Activators; D014212:Tretinoin; D008550:Melatonin