Recent advances in cyclic-adenosine monophosphate/protein kinase A signaling in ethanol-induced synaptic and behavioral alterations.
This article represents the proceedings of a symposium at the 2002 RSA Meeting in San Francisco, California, organized and co-chaired by L. Judson Chandler and Richard A. Morrisett. The presentations were (1) PKA regulates chronic ethanol-induced synaptic targeting of NMDA receptors, by L. Judson Chandler; (2) Long-lasting potentiation of GABAergic synapses in dopamine neurons after a single in vivo ethanol exposure, by Antonello Bonci; (3) The DARPP-32 cascade and regulation of the ethanol sensitivity of NMDA receptors in the nucleus accumbens, by Richard A. Morrisett; (4) and The cAMP/PKA signal transduction pathway modulates ethanol consumption and sedative effects of ethanol, by Gary S. Wand.
Chandler
L Judson
LJ
Alcohol Research Center and Department of Physiology/Neuroscience, Medical University of South Carolina, Charleston, South Carolina 29425, USA. chandj@musc.edu
Bonci
Antonello
A
Wand
Gary S
GS
Morrisett
Richard A
RA
D000818:Animals; D017868:Cyclic AMP-Dependent Protein Kinases / Q000187:drug effects* / Q000378:metabolism; D000431:Ethanol / Q000494:pharmacology*; D015398:Signal Transduction / Q000187:drug effects* / Q000502:physiology; D013569:Synapses / Q000187:drug effects* / Q000378:metabolism
D000431:Ethanol; D017868:Cyclic AMP-Dependent Protein Kinases