Endothelial function and postprandial lipemia.
It is widely recognised that post-prandial lipoproteins play a role in the development of atherosclerosis, but the mechanisms underlying this role are unclear. An attractive working hypothesis is that the pathogenetic link is endothelial dysfunction. The available data seem to corroborate this theory and recognise triggering by oxidative stress, but some of the evidence is still contradictory.
G B
GB
Vigna
C
C
Delli Gatti
R
R
Fellin
D000818:Animals; D001161:Arteriosclerosis / Q000209:etiology* / Q000503:physiopathology; D004285:Dogs; D004730:Endothelium, Vascular / Q000502:physiology*; D006801:Humans; D006949:Hyperlipidemias / Q000209:etiology* / Q000503:physiopathology; D007328:Insulin / Q000378:metabolism; D008074:Lipoproteins / Q000378:metabolism; D009569:Nitric Oxide / Q000378:metabolism*; D018384:Oxidative Stress; D019518:Postprandial Period; D012307:Risk Factors; D012680:Sensitivity and Specificity
D007328:Insulin; D008074:Lipoproteins; D009569:Nitric Oxide