[The information-wave hypothesis of a pain].
Proofs of existence in organism of two systems of nociceptive stimuli reception are submitted. The first system belongs to the neuro-endocrine system which forms painful reactions, the second one is connected with "alive matrix"--uniform system of the connective tissue which connects an environment through acupuncture points with intercellular space, and also with intracellular structures of each cell. "Alive matrix" carries out the information management of physiological processes and participates in electromagnetoreception, supervising electromagnetic parameters of an organism--"electromagnetic homeostasis". Acupuncture points carry out the role of electromagnetoreceptors. The layers of interstitial connective tissue, meridians, perform the balancing of a uniform physical field of an organism and transfer the information about its disturbances. The system of electromagnetoreception supervises the shifts in electromagnetic homeostasis of the organism. Damaging factors, physical and chemical changes in the pathological loci and fluctuations of a geomagnetic fields change a uniform physical field of an organism and initiate the compensatory responses of neuro-endocrine system. The information-wave hypothesis of a pain adequately explains effective treatment of illnesses by low intensive electromagnetic fields. The recognition of electromagnetic sensory system allows to explain the mechanism of their high medical efficiency in therapy of a pain and opens a way of new strategy, where instead of pharmacology (with its side action) safe technologies of information-wave therapy are used.
Iu P
IuP
Lymans'kyĭ
M D
MD
Kolbun
D015669:Acupuncture Points; D000818:Animals; D003238:Connective Tissue / Q000502:physiology / Q000528:radiation effects; D004574:Electromagnetic Fields; D004594:Electrophysiology; D004703:Endocrine System / Q000502:physiology / Q000528:radiation effects; D006801:Humans; D009424:Nervous System Physiological Phenomena / Q000528:radiation effects; D009619:Nociceptors / Q000502:physiology*; D010146:Pain / Q000503:physiopathology*