BCL11B rearrangements probably target T-cell neoplasia rather than acute myelocytic leukemia.
Roderick A F
RA
MacLeod
Stefan
S
Nagel
Hans G
HG
Drexler
D000818:Animals; D002454:Cell Differentiation; D019457:Chromosome Breakage; D002883:Chromosomes, Human, Pair 14 / Q000235:genetics* / Q000648:ultrastructure; D002896:Chromosomes, Human, Pair 6 / Q000235:genetics* / Q000648:ultrastructure; D004268:DNA-Binding Proteins / Q000235:genetics*; D016147:Genes, Tumor Suppressor; D019337:Hematologic Neoplasms / Q000235:genetics; D006801:Humans; D015470:Leukemia, Myeloid, Acute / Q000235:genetics*; D015458:Leukemia, T-Cell / Q000235:genetics*; D016399:Lymphoma, T-Cell / Q000235:genetics*; D008297:Male; D051379:Mice; D008875:Middle Aged; D009363:Neoplasm Proteins / Q000235:genetics*; D012097:Repressor Proteins / Q000235:genetics*; D013601:T-Lymphocytes / Q000166:cytology; D014178:Translocation, Genetic / Q000235:genetics*; D025521:Tumor Suppressor Proteins
C479740:BCL11B protein, human; D004268:DNA-Binding Proteins; D009363:Neoplasm Proteins; D012097:Repressor Proteins; D025521:Tumor Suppressor Proteins