Alterations of CaMKII after hypoxia-ischemia during brain development.
Tang
Kaixiong
K
Department of Neurology, University of Miami School of Medicine, Miami, Florida 33136, USA.
Liu
Chunli
C
Kuluz
John
J
Hu
Bingren
B
D000367:Age Factors; D000818:Animals; D001921:Brain / Q000254:growth & development / Q000473:pathology / Q000503:physiopathology*; D001923:Brain Chemistry; D054732:Calcium-Calmodulin-Dependent Protein Kinase Type 2; D017871:Calcium-Calmodulin-Dependent Protein Kinases / Q000378:metabolism*; D016923:Cell Death; D003902:Detergents / Q000737:chemistry; D004195:Disease Models, Animal; D020925:Hypoxia-Ischemia, Brain / Q000201:enzymology / Q000473:pathology / Q000503:physiopathology*; D015151:Immunoblotting; D009474:Neurons / Q000201:enzymology* / Q000473:pathology; D010766:Phosphorylation; D021381:Protein Transport; D051381:Rats; D017207:Rats, Sprague-Dawley; D012995:Solubility; D013347:Subcellular Fractions / Q000201:enzymology
D003902:Detergents; D054732:Calcium-Calmodulin-Dependent Protein Kinase Type 2; D017871:Calcium-Calmodulin-Dependent Protein Kinases