RAGE potentiates Abeta-induced perturbation of neuronal function in transgenic mice.
Arancio
Ottavio
O
Department of Psychiatry, Physiology and Neuroscience, Dementia Research Center, Nathan Kline Institute, New York University School of Medicine, NY 10032, USA.
Zhang
Hui Ping
HP
Chen
Xi
X
Lin
Chang
C
Trinchese
Fabrizio
F
Puzzo
Daniela
D
Liu
Shumin
S
Hegde
Ashok
A
Yan
Shi Fang
SF
Stern
Alan
A
Luddy
John S
JS
Lue
Lih-Fen
LF
Walker
Douglas G
DG
Roher
Alex
A
Buttini
Manuel
M
Mucke
Lennart
L
Li
Weiying
W
Schmidt
Ann Marie
AM
Kindy
Mark
M
Hyslop
Paul A
PA
Stern
David M
DM
Du Yan
Shirley Shi
SS
D000544:Alzheimer Disease / Q000378:metabolism* / Q000473:pathology*; D016564:Amyloid beta-Protein Precursor / Q000235:genetics / Q000378:metabolism*; D000818:Animals; D004195:Disease Models, Animal; D004594:Electrophysiology; D005106:Exploratory Behavior; D007249:Inflammation / Q000235:genetics / Q000473:pathology; D007858:Learning; D008297:Male; D008568:Memory; D051379:Mice; D008822:Mice, Transgenic; D016328:NF-kappa B / Q000378:metabolism; D009474:Neurons / Q000378:metabolism* / Q000473:pathology*; D000067759:Receptor for Advanced Glycation End Products; D011971:Receptors, Immunologic / Q000378:metabolism*; D016708:Synaptophysin / Q000378:metabolism
D016564:Amyloid beta-Protein Precursor; D016328:NF-kappa B; D000067759:Receptor for Advanced Glycation End Products; D011971:Receptors, Immunologic; D016708:Synaptophysin