Endogenous tissue type plasminogen activator facilitates NMDA-induced retinal damage.
Kumada
Masako
M
Department of Pharmacology, Gifu University Graduate School of Medicine, Gifu, Japan.
Niwa
Masayuki
M
Wang
Xiaodan
X
Matsuno
Hiroyuki
H
Hara
Akira
A
Mori
Hideki
H
Matsuo
Osamu
O
Yamamoto
Tetsuya
T
Kozawa
Osamu
O
D000818:Animals; D002452:Cell Count; D053938:DNA Fragmentation; D018690:Excitatory Amino Acid Agonists / Q000008:administration & dosage / Q000494:pharmacology*; D005971:Glutamates / Q000008:administration & dosage / Q000494:pharmacology; D020287:In Situ Nick-End Labeling; D007267:Injections; D007608:Kainic Acid / Q000008:administration & dosage / Q000494:pharmacology; D051379:Mice; D008810:Mice, Inbred C57BL; D018345:Mice, Knockout; D016202:N-Methylaspartate / Q000008:administration & dosage / Q000633:toxicity*; D009474:Neurons / Q000187:drug effects; D015849:Plasminogen Inactivators / Q000172:deficiency / Q000502:physiology*; D011994:Recombinant Proteins / Q000494:pharmacology; D015427:Reperfusion Injury / Q000473:pathology / Q000517:prevention & control; D012160:Retina; D012164:Retinal Diseases / Q000139:chemically induced* / Q000473:pathology; D012165:Retinal Ganglion Cells / Q000187:drug effects
D018690:Excitatory Amino Acid Agonists; D005971:Glutamates; D015849:Plasminogen Inactivators; D011994:Recombinant Proteins; D016202:N-Methylaspartate; D007608:Kainic Acid