CXCR3-dependent microglial recruitment is essential for dendrite loss after brain lesion.
Rappert
Angelika
A
Department of Cellular Neuroscience, Max Delbrück Center for Molecular Medicine, 13092 Berlin, Germany.
Bechmann
Ingo
I
Pivneva
Tatyana
T
Mahlo
Jacqueline
J
Biber
Knut
K
Nolte
Christiane
C
Kovac
Adam D
AD
Gerard
Craig
C
Boddeke
Hendrikus W G M
HW
Nitsch
Robert
R
Kettenmann
Helmut
H
D000818:Animals; D001253:Astrocytes; D019771:Axotomy; D001930:Brain Injuries / Q000473:pathology* / Q000503:physiopathology; D002452:Cell Count; D002465:Cell Movement; D054357:Chemokine CXCL10; D019743:Chemokines, CXC / Q000502:physiology; D003712:Dendrites / Q000473:pathology*; D018728:Entorhinal Cortex; D005154:Facial Nerve; D006624:Hippocampus / Q000473:pathology; D007150:Immunohistochemistry; D051379:Mice; D008810:Mice, Inbred C57BL; D018345:Mice, Knockout; D017628:Microglia / Q000502:physiology*; D019580:Perforant Pathway / Q000473:pathology*; D054367:Receptors, CXCR3; D019707:Receptors, Chemokine / Q000502:physiology*; D020133:Reverse Transcriptase Polymerase Chain Reaction; D015398:Signal Transduction / Q000502:physiology
D054357:Chemokine CXCL10; D019743:Chemokines, CXC; C515188:Cxcr3 protein, mouse; D054367:Receptors, CXCR3; D019707:Receptors, Chemokine