Pathogenesis of persistent hyperplastic primary vitreous in mice lacking the arf tumor suppressor gene.
Martin
Amy C
AC
Department of Hematology/Oncology, St. Jude Children's Research Hospital, Memphis, Tennessee 38105, USA.
Thornton
J Derek
JD
Liu
Jiewiu
J
Wang
XiaoFei
X
Zuo
Jian
J
Jablonski
Monica M
MM
Chaum
Edward
E
Zindy
Frederique
F
Skapek
Stephen X
SX
D000818:Animals; D019941:Cyclin-Dependent Kinase Inhibitor p16 / Q000235:genetics / Q000378:metabolism; D005124:Eye Abnormalities / Q000209:etiology* / Q000378:metabolism / Q000473:pathology; D017353:Gene Deletion; D005786:Gene Expression Regulation; D005838:Genotype; D049452:Green Fluorescent Proteins; D006965:Hyperplasia; D007150:Immunohistochemistry; D017403:In Situ Hybridization; D008164:Luminescent Proteins; D051379:Mice; D008810:Mice, Inbred C57BL; D018345:Mice, Knockout; D009389:Neovascularization, Pathologic / Q000209:etiology / Q000378:metabolism / Q000473:pathology; D009887:Ophthalmoscopy; D012333:RNA, Messenger / Q000378:metabolism; D020133:Reverse Transcriptase Polymerase Chain Reaction; D025803:Tumor Suppressor Protein p14ARF / Q000235:genetics* / Q000378:metabolism; D014822:Vitreous Body / Q000002:abnormalities* / Q000098:blood supply* / Q000473:pathology
C417743:Cdkn2a protein, mouse; D019941:Cyclin-Dependent Kinase Inhibitor p16; D008164:Luminescent Proteins; D012333:RNA, Messenger; D025803:Tumor Suppressor Protein p14ARF; D049452:Green Fluorescent Proteins