High glucose concentrations stimulate human monocyte sodium/hydrogen exchanger activity and modulate atherosclerosis-related functions.
Koliakos
G
G
Department of Biological Chemistry, Medical School, Aristotle University of Thessaloniki, PO Box 17034, 54124 Thessaloniki, Greece. koliakos@med.auth.gr
Zolota
Z
Z
Paletas
K
K
Kaloyianni
M
M
D001161:Arteriosclerosis / Q000378:metabolism*; D018955:CD36 Antigens / Q000378:metabolism*; D027682:Cation Transport Proteins / Q000378:metabolism*; D002448:Cell Adhesion / Q000187:drug effects; D002465:Cell Movement / Q000187:drug effects; D002478:Cells, Cultured; D004789:Enzyme Activation; D005947:Glucose / Q000378:metabolism* / Q000494:pharmacology; D006801:Humans; D007797:Laminin / Q000378:metabolism*; D008565:Membrane Proteins / Q000378:metabolism*; D009000:Monocytes / Q000187:drug effects / Q000378:metabolism*; D000074082:Sodium-Hydrogen Exchanger 1; D017923:Sodium-Hydrogen Exchangers / Q000378:metabolism*
D018955:CD36 Antigens; D027682:Cation Transport Proteins; D007797:Laminin; D008565:Membrane Proteins; C490998:SLC9A1 protein, human; D000074082:Sodium-Hydrogen Exchanger 1; D017923:Sodium-Hydrogen Exchangers; D005947:Glucose