Inhibition of caspase-3 activation and apoptosis is involved in 3-nitropropionic acid-induced ischemic tolerance to transient focal cerebral ischemia in rats.
Zhu
Hong-Can
HC
Department of Neurology, The First Affiliated Hospital, Zhengzhou University, Zhengzhou 450052, China. zhc660407@hotmail.com
Gao
Xia-Qun
XQ
Xing
Ying
Y
Sun
Sheng-Gang
SG
Li
Hong-Ge
HG
Wang
Yun-Fu
YF
D000818:Animals; D017209:Apoptosis / Q000502:physiology*; D001921:Brain / Q000166:cytology / Q000187:drug effects* / Q000378:metabolism / Q000473:pathology; D053148:Caspase 3; D061945:Caspase Inhibitors; D020169:Caspases / Q000378:metabolism*; D053938:DNA Fragmentation; D004789:Enzyme Activation; D020287:In Situ Nick-End Labeling; D002546:Ischemic Attack, Transient / Q000378:metabolism* / Q000473:pathology; D019194:Ischemic Preconditioning; D008297:Male; D009498:Neurotoxins / Q000494:pharmacology; D009574:Nitro Compounds; D011422:Propionates / Q000494:pharmacology*; D051381:Rats; D017207:Rats, Sprague-Dawley
D061945:Caspase Inhibitors; D009498:Neurotoxins; D009574:Nitro Compounds; D011422:Propionates; C505410:Casp3 protein, rat; D053148:Caspase 3; D020169:Caspases; C015392:3-nitropropionic acid