Plaque neovascularization is increased in ruptured atherosclerotic lesions of human aorta: implications for plaque vulnerability.
Moreno
Pedro R
PR
Cardiovascular Institute, Mount Sinai School of Medicine, One Gustave L. Levy Place, Box 1030, New York, NY 10029, USA. pedro.moreno@msnyuhealth.org.
Purushothaman
K Raman
KR
Fuster
Valentin
V
Echeverri
Dario
D
Truszczynska
Helena
H
Sharma
Samin K
SK
Badimon
Juan J
JJ
O'Connor
William N
WN
D000328:Adult; D000368:Aged; D000369:Aged, 80 and over; D018952:Antigens, CD34 / Q000032:analysis; D001012:Aorta, Abdominal / Q000473:pathology; D001018:Aortic Diseases / Q000150:complications / Q000473:pathology*; D001161:Arteriosclerosis / Q000150:complications / Q000473:pathology*; D003097:Collateral Circulation; D005355:Fibrosis; D006470:Hemorrhage / Q000209:etiology / Q000473:pathology; D006801:Humans; D008264:Macrophages / Q000473:pathology; D008297:Male; D008875:Middle Aged; D032389:Myocytes, Smooth Muscle / Q000473:pathology; D012422:Rupture, Spontaneous; D016176:T-Lymphocyte Subsets / Q000473:pathology; D013927:Thrombosis / Q000209:etiology / Q000473:pathology; D017540:Tunica Media / Q000473:pathology; D014651:Vasa Vasorum / Q000473:pathology*; D014657:Vasculitis / Q000209:etiology / Q000473:pathology
D018952:Antigens, CD34