title: Asymmetric Nodal expression in the mouse is governed by the combinatorial activities of two distinct regulatory elements.
authors.affiliation: Department of Molecular and Cellular Biology, Harvard University, 16 Divinity Avenue, Cambridge, MA 02138, USA. stephanie.vincent@ens.lyon.fr
authors.forename: Stéphane D
authors.initials: SD
authors.lastname: Vincent
authors.forename: Dominic P
authors.initials: DP
authors.lastname: Norris
authors.forename: J Ann
authors.initials: JA
authors.lastname: Le Good
authors.forename: Daniel B
authors.initials: DB
authors.lastname: Constam
authors.forename: Elizabeth J
authors.initials: EJ
authors.lastname: Robertson
mesh_terms: D000483:Alleles; D000818:Animals; D001483:Base Sequence; D019521:Body Patterning / Q000235:genetics*; D002874:Chromosome Mapping; D004622:Embryo, Mammalian / Q000002:abnormalities; D004742:Enhancer Elements, Genetic / Q000235:genetics*; D018507:Gene Expression Regulation, Developmental / Q000235:genetics*; D017403:In Situ Hybridization; D051379:Mice; D008822:Mice, Transgenic; D008969:Molecular Sequence Data; D055457:Nodal Protein; D012333:RNA, Messenger / Q000032:analysis / Q000378:metabolism; D017384:Sequence Deletion / Q000235:genetics; D016212:Transforming Growth Factor beta / Q000096:biosynthesis / Q000235:genetics*; D014781:Viscera / Q000196:embryology
chemical_list: D055457:Nodal Protein; C526008:Nodal protein, mouse; D012333:RNA, Messenger; D016212:Transforming Growth Factor beta