Selective inactivation or reconstitution of adenosine A2A receptors in bone marrow cells reveals their significant contribution to the development of ischemic brain injury.
Yu
Liqun
L
Department of Neurology, Boston University School of Medicine, 715 Albany Street, C329, Boston, Massachusetts 02118, USA.
Huang
Zhihong
Z
Mariani
Juliana
J
Wang
Yumei
Y
Moskowitz
Michael
M
Chen
Jiang-Fan
JF
D000818:Animals; D001854:Bone Marrow Cells / Q000378:metabolism*; D016026:Bone Marrow Transplantation; D001921:Brain / Q000098:blood supply* / Q000378:metabolism; D002678:Chimera; D016207:Cytokines / Q000378:metabolism; D007150:Immunohistochemistry; D002546:Ischemic Attack, Transient / Q000378:metabolism / Q000503:physiopathology*; D008099:Liver / Q000098:blood supply* / Q000378:metabolism; D051379:Mice; D018345:Mice, Knockout; D009504:Neutrophils / Q000378:metabolism; D016133:Polymerase Chain Reaction; D043705:Receptor, Adenosine A2A / Q000378:metabolism*
D016207:Cytokines; D043705:Receptor, Adenosine A2A