title: Selective inactivation or reconstitution of adenosine A2A receptors in bone marrow cells reveals their significant contribution to the development of ischemic brain injury.
authors.affiliation: Department of Neurology, Boston University School of Medicine, 715 Albany Street, C329, Boston, Massachusetts 02118, USA.
authors.forename: Liqun
authors.initials: L
authors.lastname: Yu
authors.forename: Zhihong
authors.initials: Z
authors.lastname: Huang
authors.forename: Juliana
authors.initials: J
authors.lastname: Mariani
authors.forename: Yumei
authors.initials: Y
authors.lastname: Wang
authors.forename: Michael
authors.initials: M
authors.lastname: Moskowitz
authors.forename: Jiang-Fan
authors.initials: JF
authors.lastname: Chen
mesh_terms: D000818:Animals; D001854:Bone Marrow Cells / Q000378:metabolism*; D016026:Bone Marrow Transplantation; D001921:Brain / Q000098:blood supply* / Q000378:metabolism; D002678:Chimera; D016207:Cytokines / Q000378:metabolism; D007150:Immunohistochemistry; D002546:Ischemic Attack, Transient / Q000378:metabolism / Q000503:physiopathology*; D008099:Liver / Q000098:blood supply* / Q000378:metabolism; D051379:Mice; D018345:Mice, Knockout; D009504:Neutrophils / Q000378:metabolism; D016133:Polymerase Chain Reaction; D043705:Receptor, Adenosine A2A / Q000378:metabolism*
chemical_list: D016207:Cytokines; D043705:Receptor, Adenosine A2A