Persistent infection with primate foamy virus type 1 increases human immunodeficiency virus type 1 cell binding via a Bet-independent mechanism.
Schiffer
Cecile
C
Laboratoire d'Immunologie Cellulaire et Immunopathologie de l'EPHE, EMI-0013, Institut Universitaire d'Hématologie, Centre Hayem, Hôpital Saint Louis, 1 avenue Claude Vellefaux, 75475 Paris CEDEX 10, France.
Lecellier
Charles-Henri
CH
Mannioui
Abdelkrim
A
Felix
Nathalie
N
Nelson
Elisabeth
E
Lehmann-Che
Jacqueline
J
Giron
Marie-Louise
ML
Gluckman
Jean Claude
JC
Saib
Ali
A
Canque
Bruno
B
D000818:Animals; D002908:Chronic Disease; D015658:HIV Infections / Q000150:complications* / Q000821:virology; D015497:HIV-1 / Q000378:metabolism / Q000472:pathogenicity / Q000502:physiology*; D006801:Humans; D012192:Retroviridae Infections / Q000150:complications* / Q000821:virology; D012191:Retroviridae Proteins / Q000378:metabolism*; D016092:Spumavirus / Q000472:pathogenicity*; D013601:T-Lymphocytes / Q000821:virology*; D014779:Virus Replication
D012191:Retroviridae Proteins; C080099:bet protein, human foamy virus