Increases in oxidative stress in the progeny of X-irradiated cells.
Rugo
Rebecca E
RE
Department of Cancer Cell Biology, Harvard School of Public Health, Boston, Massachusetts 02115, USA.
Schiestl
Robert H
RH
D017209:Apoptosis; D015153:Blotting, Western; D002453:Cell Cycle; D016923:Cell Death; D002455:Cell Division; D002460:Cell Line; D002470:Cell Survival; D004249:DNA Damage; D004307:Dose-Response Relationship, Radiation; D005434:Flow Cytometry; D005452:Fluoresceins / Q000494:pharmacology; D016193:G1 Phase; D016158:Genes, p53; D006419:Heme Oxygenase (Decyclizing) / Q000378:metabolism; D051547:Heme Oxygenase-1; D006801:Humans; D008214:Lymphocytes / Q000528:radiation effects*; D020935:MAP Kinase Signaling System; D008565:Membrane Proteins; D020928:Mitogen-Activated Protein Kinases / Q000378:metabolism; D018384:Oxidative Stress; D010641:Phenotype; D017382:Reactive Oxygen Species; D015398:Signal Transduction; D013997:Time Factors; D014162:Transfection; D014965:X-Rays
D005452:Fluoresceins; D008565:Membrane Proteins; D017382:Reactive Oxygen Species; C037631:2',7'-dichlorofluorescein; C496541:HMOX1 protein, human; D006419:Heme Oxygenase (Decyclizing); D051547:Heme Oxygenase-1; D020928:Mitogen-Activated Protein Kinases