Chagas' disease parasite promotes neuron survival and differentiation through TrkA nerve growth factor receptor.
Chuenkova
Marina V
MV
Parasitology Research Center, Department of Pathology, Tufts University School of Medicine, Boston, Massachusetts 02111, USA.
PereiraPerrin
Mercio
M
D000818:Animals; D001369:Axons / Q000187:drug effects; D001667:Binding, Competitive; D002454:Cell Differentiation / Q000187:drug effects; D002470:Cell Survival / Q000187:drug effects; D002478:Cells, Cultured; D003712:Dendrites / Q000187:drug effects; D006624:Hippocampus / Q000166:cytology; D006790:Host-Parasite Interactions; D008024:Ligands; D018716:Molecular Mimicry; D009439:Neuraminidase / Q000737:chemistry / Q000378:metabolism / Q000494:pharmacology*; D009474:Neurons / Q000187:drug effects* / Q000469:parasitology* / Q000502:physiology; D016716:PC12 Cells; D010766:Phosphorylation / Q000187:drug effects; D011233:Precipitin Tests; D015800:Protozoan Proteins / Q000737:chemistry / Q000378:metabolism / Q000494:pharmacology*; D051381:Rats; D020917:Receptor, trkA / Q000737:chemistry / Q000187:drug effects / Q000378:metabolism*; D014349:Trypanosoma cruzi / Q000201:enzymology*
D008024:Ligands; D015800:Protozoan Proteins; D020917:Receptor, trkA; D009439:Neuraminidase