title: [Chlamydia pneumoniae and cardiovascular diseases].
abstract: The theories formulated to explain atherogenesis evolved from vessel wall lipid to endothelial dysfunction with vascular wall remodelling. Chlamydia pneumoniae has been demonstrated to play an important role in the development of inflammatory and immunological process and can initiate atherogenesis. This pathogen is found in atherosclerotic plaques, which can induce complex activation of local inflammatory and immunological events and lead to the development of unstable plaque, vascular remodelling and vessel lumen constriction.
authors.affiliation: II Klinika Kardiologii, Instytutu Kardiologii, Collegium Medicum, Uniwersytetu Jagiellońskiego w Krakowie.
authors.forename: Renata
authors.initials: R
authors.lastname: Rajtar
authors.forename: Małgorzata
authors.initials: M
authors.lastname: Malczewska-Malec
authors.forename: Małgorzata
authors.initials: M
authors.lastname: Kloch
authors.forename: Władysława
authors.initials: W
authors.lastname: Kolasińska-Kloch
mesh_terms: D000818:Animals; D002690:Chlamydia Infections / Q000150:complications* / Q000276:immunology / Q000473:pathology; D016993:Chlamydophila pneumoniae / Q000302:isolation & purification*; D003324:Coronary Artery Disease / Q000382:microbiology* / Q000473:pathology; D003331:Coronary Vessels / Q000382:microbiology* / Q000473:pathology; D004730:Endothelium, Vascular / Q000382:microbiology / Q000473:pathology; D006801:Humans; D012307:Risk Factors