title: Fingering IL-12 with apoptotic cells.
abstract: Exposure of phosphatidylserine on the surface of apoptotic cells contributes to their removal in situ by quiet, noninflammatory, and nonimmunogenic mechanisms. In this issue of Immunity, describe a completely new mechanism for this effect that involves a novel zinc finger nuclear factor that inhibits IL-12 production.
authors.affiliation: National Jewish Medical and Research Center, 1400 Jackson Street, Denver, CO 80206, USA.
authors.forename: Peter M
authors.initials: PM
authors.lastname: Henson
mesh_terms: D017209:Apoptosis; D006801:Humans; D018664:Interleukin-12 / Q000235:genetics*; D010766:Phosphorylation; D012097:Repressor Proteins / Q000502:physiology*; D016335:Zinc Fingers
chemical_list: C072278:GCFC2 protein, human; D012097:Repressor Proteins; D018664:Interleukin-12