[ATP and neuropathic pain].
Inoue
Kazuhide
K
Division of Biosignaling, National Institute of Health Sciences, 1-18-1 Kamiyoga, Setagaya, Tokyo 158-8501, Japan.
Tsuda
Makoto
M
Koizumi
Shuichi
S
D000255:Adenosine Triphosphate / Q000502:physiology*; D000818:Animals; D015195:Drug Design; D015870:Gene Expression; D006801:Humans; D017628:Microglia / Q000378:metabolism; D009437:Neuralgia / Q000235:genetics*; D010148:Intractable Pain / Q000235:genetics*; D018048:Receptors, Purinergic P2 / Q000235:genetics / Q000502:physiology*; D058476:Receptors, Purinergic P2X2; D058477:Receptors, Purinergic P2X3; D058848:Receptors, Purinergic P2X4; D015398:Signal Transduction / Q000235:genetics / Q000502:physiology; D014544:Uridine Triphosphate / Q000494:pharmacology / Q000502:physiology
C546401:P2RX2 protein, human; C546407:P2RX3 protein, human; C548226:P2RX4 protein, human; D018048:Receptors, Purinergic P2; D058476:Receptors, Purinergic P2X2; D058477:Receptors, Purinergic P2X3; D058848:Receptors, Purinergic P2X4; D000255:Adenosine Triphosphate; D014544:Uridine Triphosphate