title: Avoiding the effect of linked genes is crucial to elucidate the role of Apcs in autoimmunity.
authors.forename: Toshio
authors.initials: T
authors.lastname: Tamaoki
authors.forename: Hideo
authors.initials: H
authors.lastname: Tezuka
authors.forename: Yoshiie
authors.initials: Y
authors.lastname: Okada
authors.forename: Sadahiro
authors.initials: S
authors.lastname: Ito
authors.forename: Hiroki
authors.initials: H
authors.lastname: Shimura
authors.forename: Mihoko
authors.initials: M
authors.lastname: Sakamoto
authors.forename: Toyoshi
authors.initials: T
authors.lastname: Endo
authors.forename: Yukio
authors.initials: Y
authors.lastname: Ozaki
authors.forename: Shigenobu
authors.initials: S
authors.lastname: Kanba
authors.forename: Shuichiro
authors.initials: S
authors.lastname: Maeda
mesh_terms: D000818:Animals; D015551:Autoimmunity / Q000235:genetics* / Q000502:physiology; D008040:Genetic Linkage; D047908:Intracellular Signaling Peptides and Proteins / Q000235:genetics / Q000378:metabolism; D051379:Mice; D008810:Mice, Inbred C57BL; D010750:Phosphoproteins / Q000235:genetics / Q000378:metabolism; D012333:RNA, Messenger / Q000378:metabolism; D000683:Serum Amyloid P-Component / Q000235:genetics* / Q000378:metabolism
chemical_list: C092537:Ifi202b protein, mouse; D047908:Intracellular Signaling Peptides and Proteins; D010750:Phosphoproteins; D012333:RNA, Messenger; D000683:Serum Amyloid P-Component