title: Stress response and apoptosis in pro- and antiinflammatory macrophages.
abstract: We showed that stress response and apoptosis in macrophages depend on the phenotype of their secretory activity and specific biological and physical characteristics of the factor inducing stress-response or apoptosis.
authors.affiliation: Institute of General Pathology and Pathophysiology, Russian Academy of Medical Sciences, Moscow. igor.malyshev@mtu-net.ru
authors.forename: I Yu
authors.initials: IY
authors.lastname: Malyshev
authors.forename: S V
authors.initials: SV
authors.lastname: Kruglov
authors.forename: L Yu
authors.initials: LY
authors.lastname: Bakhtina
authors.forename: E V
authors.initials: EV
authors.lastname: Malysheva
authors.forename: M
authors.initials: M
authors.lastname: Zubin
authors.forename: M
authors.initials: M
authors.lastname: Norkin
mesh_terms: D000818:Animals; D017209:Apoptosis / Q000502:physiology*; D004268:DNA-Binding Proteins / Q000378:metabolism; D018840:HSP70 Heat-Shock Proteins / Q000378:metabolism; D000076249:Heat Shock Transcription Factors; D007249:Inflammation / Q000503:physiopathology*; D008070:Lipopolysaccharides / Q000494:pharmacology; D017737:Macrophages, Peritoneal / Q000276:immunology* / Q000502:physiology*; D008297:Male; D051379:Mice; D010641:Phenotype; D014157:Transcription Factors / Q000378:metabolism
chemical_list: D004268:DNA-Binding Proteins; D018840:HSP70 Heat-Shock Proteins; D000076249:Heat Shock Transcription Factors; C490017:Hsf1 protein, mouse; D008070:Lipopolysaccharides; D014157:Transcription Factors