Polyglutamine repeat length in the NCOA3 does not affect risk in familial breast cancer.
Wilkening
Stefan
S
Department of Molecular Genetic Epidemiology, German Cancer Research Center, Im Neuenheimer Feld 580, 69120 Heidelberg, Germany. stefan_wilkening@web.de
Burwinkel
Barbara
B
Grzybowska
Ewa
E
Klaes
Rüdiger
R
Pamula
Jolanta
J
Pekala
Wioletta
W
Zientek
Helena
H
Hemminki
Kari
K
Försti
Asta
A
D000123:Acetyltransferases; D000328:Adult; D001943:Breast Neoplasms / Q000235:genetics*; D016022:Case-Control Studies; D016009:Chi-Square Distribution; D005260:Female; D019398:Genes, BRCA1; D024522:Genes, BRCA2; D020022:Genetic Predisposition to Disease; D018095:Germ-Line Mutation; D005858:Germany; D051548:Histone Acetyltransferases; D006801:Humans; D008875:Middle Aged; D056921:Nuclear Receptor Coactivator 3; D015513:Oncogene Proteins; D011044:Poland; D011099:Polyglutamic Acid / Q000235:genetics*; D012307:Risk Factors; D015534:Trans-Activators / Q000235:genetics*
D015513:Oncogene Proteins; D015534:Trans-Activators; D011099:Polyglutamic Acid; D000123:Acetyltransferases; D051548:Histone Acetyltransferases; C538866:NCOA3 protein, human; D056921:Nuclear Receptor Coactivator 3