Stress response and apoptosis in pro- and antiinflammatory macrophages.
We showed that stress response and apoptosis in macrophages depend on the phenotype of their secretory activity and specific biological and physical characteristics of the factor inducing stress-response or apoptosis.
Malyshev
I Yu
IY
Institute of General Pathology and Pathophysiology, Russian Academy of Medical Sciences, Moscow. igor.malyshev@mtu-net.ru
Kruglov
S V
SV
Bakhtina
L Yu
LY
Malysheva
E V
EV
Zubin
M
M
Norkin
M
M
D000818:Animals; D017209:Apoptosis / Q000502:physiology*; D004268:DNA-Binding Proteins / Q000378:metabolism; D018840:HSP70 Heat-Shock Proteins / Q000378:metabolism; D000076249:Heat Shock Transcription Factors; D007249:Inflammation / Q000503:physiopathology*; D008070:Lipopolysaccharides / Q000494:pharmacology; D017737:Macrophages, Peritoneal / Q000276:immunology* / Q000502:physiology*; D008297:Male; D051379:Mice; D010641:Phenotype; D014157:Transcription Factors / Q000378:metabolism
D004268:DNA-Binding Proteins; D018840:HSP70 Heat-Shock Proteins; D000076249:Heat Shock Transcription Factors; C490017:Hsf1 protein, mouse; D008070:Lipopolysaccharides; D014157:Transcription Factors