A topodiagnostic investigation on body lateropulsion in medullary infarcts.
Body lateropulsion may occur without signs of vestibular dysfunction and vestibular nucleus involvement. The authors examined 10 such patients with three-dimensional brainstem mapping. Body lateropulsion without limb ataxia reflected an impairment of vestibulospinal postural control caused by a lesion of the descending lateral vestibulospinal tract, whereas body lateropulsion with limb ataxia was probably the consequence of impaired or absent proprioceptive information caused by a lesion of the ascending dorsal spino-cerebellar tract.
Thömke
F
F
Department of Neurology, University of Mainz, Langenbeckstr. 1, D-55101 Mainz, Germany. thoemke@neurologie.klinik.uni-mainz.de
Marx
J J
JJ
Iannetti
G D
GD
Cruccu
G
G
Fitzek
S
S
Urban
P P
PP
Stoeter
P
P
Dieterich
M
M
Hopf
H C
HC
D001259:Ataxia / Q000209:etiology*; D001931:Brain Mapping; D003680:Deglutition Disorders / Q000209:etiology; D003937:Diagnosis, Differential; D038524:Diffusion Magnetic Resonance Imaging; D006732:Horner Syndrome / Q000209:etiology; D006801:Humans; D014854:Lateral Medullary Syndrome / Q000175:diagnosis / Q000209:etiology / Q000503:physiopathology*; D011446:Prospective Studies; D012678:Sensation Disorders / Q000209:etiology; D014715:Vertebrobasilar Insufficiency / Q000150:complications / Q000503:physiopathology; D015837:Vestibular Diseases / Q000175:diagnosis