title: Pelvic floor trauma in childbirth - myth or reality?
abstract: The issue of traumatic damage to the pelvic floor in childbirth is attracting more and more attention amongst obstetric caregivers and laypersons alike. This is partly due to the fact that elective Caesarean section, as a potentially preventative intervention, is increasingly available and perceived as safe. As there are a multitude of emotive issues involved, including health economics and the relative roles of healthcare providers, the discussion surrounding pelvic floor trauma in childbirth has not always been completely rational. However, after 25 years of urogynaecological research in this field it should be possible to determine whether pelvic floor trauma in childbirth is myth or reality. On reviewing the available evidence, it appears that there are sufficient grounds to assume that vaginal delivery (or even the attempt at vaginal delivery) can cause damage to the pudendal nerve, the caudal aspects of the levator ani muscle, fascial pelvic organ supports and the external and internal anal sphincter. Risk factors for such damage have been defined and variously include operative vaginal delivery, a long second stage, and macrosomia. It is much less clear, however, whether such trauma is clinically relevant, and how important it is in the aetiology of pelvic floor morbidity later in life.
authors.affiliation: University of Sydney, Western Clinical School, Penrith, New South Wales, Australia. hpdietz@bigpond.com
authors.forename: H P
authors.initials: HP
authors.lastname: Dietz
authors.forename: Lore
authors.initials: L
authors.lastname: Schierlitz
mesh_terms: D036861:Delivery, Obstetric / Q000009:adverse effects*; D005242:Fecal Incontinence / Q000209:etiology; D005260:Female; D006801:Humans; D018482:Muscle, Skeletal / Q000293:injuries; D007744:Obstetric Labor Complications / Q000503:physiopathology; D036801:Parturition; D017773:Pelvic Floor / Q000293:injuries*; D010388:Pelvis / Q000294:innervation; D011247:Pregnancy; D014549:Urinary Incontinence / Q000209:etiology