title: [Calcitonin and renal mineral reabsorption].
abstract: Calcitonin and its receptor are well known for their ability to regulate osteoclast-mediated bone resorption. Calcitonin has been suggested to be a renal Ca(2+) and Mg(2+)-conserving hormone and may share similar signaling mechanisms with PTH. This review describes the characteristics of renal Ca(2+), Mg(2+) and Pi transport pathway and the physiological roles of calcitonin in renal mineral reabsorption.
authors.affiliation: Department of Molecular Nutrition, Institute of Health biosciences, The University of Tokushima Graduate School.
authors.forename: Ken-ichi
authors.initials: K
authors.lastname: Miyamoto
authors.forename: Mikiko
authors.initials: M
authors.lastname: Ito
authors.forename: Hiroko
authors.initials: H
authors.lastname: Segawa
mesh_terms: D002116:Calcitonin / Q000502:physiology*; D002118:Calcium / Q000378:metabolism*; D006801:Humans; D007684:Kidney Tubules / Q000502:physiology*; D008274:Magnesium / Q000378:metabolism*
chemical_list: D002116:Calcitonin; D008274:Magnesium; D002118:Calcium