Pseudomonas aeruginosa OxyR is required for full virulence in rodent and insect models of infection and for resistance to human neutrophils.
Lau
Gee W
GW
Department of Internal Medicine, University of Cincinnati College of Medicine, Cincinnati, OH 45267-0524, USA.
Britigan
Bradley E
BE
Hassett
Daniel J
DJ
D000818:Animals; D001770:Blood Bactericidal Activity; D004268:DNA-Binding Proteins / Q000502:physiology*; D004331:Drosophila melanogaster; D006801:Humans; D051379:Mice; D009504:Neutrophils / Q000276:immunology*; D018410:Pneumonia, Bacterial / Q000209:etiology; D011552:Pseudomonas Infections / Q000209:etiology*; D011550:Pseudomonas aeruginosa / Q000472:pathogenicity*; D012097:Repressor Proteins / Q000502:physiology*; D014157:Transcription Factors / Q000502:physiology*; D014774:Virulence; D014946:Wound Infection / Q000209:etiology
D004268:DNA-Binding Proteins; D012097:Repressor Proteins; D014157:Transcription Factors