[Mechanism of progression of atherosclerosis in patients with impaired glucose tolerance (IGT)].
Watada
Hirotaka
H
Department of Medicine, Metabolism and Endocrinology, Juntendo University School of Medicine.
Nomiyama
Takashi
T
Kawamori
Ryuzo
R
D052242:Adiponectin; D000818:Animals; D001161:Arteriosclerosis / Q000209:etiology* / Q000473:pathology; D002448:Cell Adhesion; D016207:Cytokines / Q000378:metabolism; D018450:Disease Progression; D042783:Endothelial Cells / Q000473:pathology; D005230:Fatty Acids, Nonesterified / Q000009:adverse effects; D005487:Foam Cells / Q000378:metabolism; D018149:Glucose Intolerance / Q000150:complications* / Q000503:physiopathology; D006801:Humans; D006943:Hyperglycemia / Q000150:complications; D006949:Hyperlipidemias / Q000150:complications; D007328:Insulin / Q000378:metabolism; D007333:Insulin Resistance; D000078790:Insulin Secretion; D036341:Intercellular Signaling Peptides and Proteins / Q000502:physiology; D008019:Life Style; D018384:Oxidative Stress
D052242:Adiponectin; D016207:Cytokines; D005230:Fatty Acids, Nonesterified; D007328:Insulin; D036341:Intercellular Signaling Peptides and Proteins