[Interleukin-18 in pathogenesis of rheumatoid arthritis].
Okamura
Haruki
H
Department of Laboratory of Host Defenses, Institute for Advanced Medical Science, Hyogo College of Medicine.
Kashiwamura
Shin-ichiro
S
Ueda
Haruyasu
H
D000818:Animals; D001172:Arthritis, Rheumatoid / Q000209:etiology* / Q000628:therapy; D018414:CD8-Positive T-Lymphocytes / Q000276:immunology; D016207:Cytokines / Q000096:biosynthesis; D003602:Cytotoxicity, Immunologic; D053222:Fas Ligand Protein; D006801:Humans; D018836:Inflammation Mediators; D020382:Interleukin-18 / Q000096:biosynthesis / Q000502:physiology*; D053727:Interleukin-18 Receptor alpha Subunit; D007694:Killer Cells, Natural / Q000276:immunology; D008562:Membrane Glycoproteins; D018123:Receptors, Interleukin / Q000502:physiology; D053724:Receptors, Interleukin-18; D015398:Signal Transduction / Q000235:genetics / Q000502:physiology; D018417:Th1 Cells / Q000276:immunology; D018418:Th2 Cells / Q000276:immunology
D016207:Cytokines; C505661:FASLG protein, human; D053222:Fas Ligand Protein; C508572:IL18R1 protein, human; D018836:Inflammation Mediators; D020382:Interleukin-18; D053727:Interleukin-18 Receptor alpha Subunit; D008562:Membrane Glycoproteins; D018123:Receptors, Interleukin; D053724:Receptors, Interleukin-18