[Disorder of glucose metabolism in skeletal muscle].
Horike
Nanao
N
Department of Physiological Chemistry and Metabolism, Graduate School of Medicine, University of Tokyo.
Asano
Tomoichiro
T
D055372:AMP-Activated Protein Kinases; D000818:Animals; D005947:Glucose / Q000378:metabolism*; D018149:Glucose Intolerance / Q000209:etiology*; D051275:Glucose Transporter Type 4; D006595:Hexosamines / Q000096:biosynthesis; D006801:Humans; D007328:Insulin / Q000502:physiology; D007333:Insulin Resistance; D009004:Monosaccharide Transport Proteins / Q000502:physiology; D009097:Multienzyme Complexes / Q000502:physiology; D009119:Muscle Contraction / Q000235:genetics / Q000502:physiology; D009124:Muscle Proteins / Q000502:physiology; D018482:Muscle, Skeletal / Q000378:metabolism*; D019869:Phosphatidylinositol 3-Kinases / Q000502:physiology; D017346:Protein Serine-Threonine Kinases / Q000502:physiology; D015398:Signal Transduction / Q000235:genetics
D055372:AMP-Activated Protein Kinases; D005947:Glucose; D051275:Glucose Transporter Type 4; D006595:Hexosamines; D007328:Insulin; D009004:Monosaccharide Transport Proteins; D009097:Multienzyme Complexes; D009124:Muscle Proteins; D019869:Phosphatidylinositol 3-Kinases; D017346:Protein Serine-Threonine Kinases; C495692:SLC2A4 protein, human