[Physiological and pathological roles of apoptosis].
Suda
Takashi
T
Center for the Development of Molecular Target Drugs, Cancer Research Institute, Kanazawa University.
D000818:Animals; D017209:Apoptosis / Q000235:genetics / Q000276:immunology / Q000502:physiology*; D053477:Apoptotic Protease-Activating Factor 1; D001402:B-Lymphocytes / Q000502:physiology*; D001773:Blood Cells / Q000502:physiology; D023201:CD40 Ligand / Q000502:physiology; D020169:Caspases / Q000502:physiology; D016207:Cytokines / Q000378:metabolism; D053222:Fas Ligand Protein; D006801:Humans; D007249:Inflammation / Q000209:etiology; D018836:Inflammation Mediators / Q000378:metabolism; D008562:Membrane Glycoproteins / Q000502:physiology; D016328:NF-kappa B / Q000378:metabolism; D009369:Neoplasms / Q000209:etiology; D011506:Proteins / Q000502:physiology; D013601:T-Lymphocytes / Q000502:physiology*
C506592:APAF1 protein, human; D053477:Apoptotic Protease-Activating Factor 1; D016207:Cytokines; C505661:FASLG protein, human; D053222:Fas Ligand Protein; D018836:Inflammation Mediators; D008562:Membrane Glycoproteins; D016328:NF-kappa B; D011506:Proteins; D023201:CD40 Ligand; D020169:Caspases