title: Hemidystonia precipitated by acute pontine infarct.
abstract: Hemidystonia is frequently due to an underlying structural lesion in the basal ganglia and thalamus. It has been suggested that a preserved corticospinal tract may be required for hemidystonia to manifest. We provide the first report of a patient who presented with rapid-onset hemidystonia precipitated by an acute pontine infarct demonstrated on diffusion-weighted magnetic resonance imaging. Acute dysregulation of pallidal efferents to the pedunculopontine and/or pontine afferents to the thalamus may precipitate hemidystonia.
authors.affiliation: Department of Neurology, Singapore General Hospital, Singapore. gnrtek@sgh.com.sg
authors.forename: E K
authors.initials: EK
authors.lastname: Tan
authors.forename: L L
authors.initials: LL
authors.lastname: Chan
authors.forename: A P
authors.initials: AP
authors.lastname: Auchus
mesh_terms: D000328:Adult; D020520:Brain Infarction / Q000473:pathology / Q000503:physiopathology*; D038524:Diffusion Magnetic Resonance Imaging / Q000379:methods; D004421:Dystonia / Q000209:etiology* / Q000473:pathology; D006801:Humans; D008297:Male; D011149:Pons / Q000473:pathology / Q000503:physiopathology*