[Study of CTnI immunohistochemistry on acute myocardial infarction in rabbits].
OBJECTIVE
To investigate the signification of CTnI in acute myocardial infarction.


METHODS
The model of myocardial infarction was established by ligating the left ventricle branch. Immunohistochemistry and image analysis were used to detect the change of CTnI in the model, and The sensitivity of immunohistochemistry and HE coloration were also compared.


RESULTS
The acute myocardial infarction tissue showed obvious depletion of CTnI, there was no characterization of myocardial infarction in HE coloration.


CONCLUSIONS
CTnI is sensitive to diagnosis of acute myocardial infarction.
Jia
Jian-zhang
JZ
Department of Forensic Medicine, Shanghai Medical College, Fudan University, Shanghai, China.
Zhao
Zi-qin
ZQ
Gu
Yun-ju
YJ
D000818:Animals; D000911:Antibodies, Monoclonal; D004195:Disease Models, Animal; D005260:Female; D007150:Immunohistochemistry; D008297:Male; D009203:Myocardial Infarction / Q000175:diagnosis* / Q000378:metabolism*; D017202:Myocardial Ischemia / Q000378:metabolism / Q000473:pathology; D009206:Myocardium / Q000378:metabolism* / Q000473:pathology; D011817:Rabbits; D012680:Sensitivity and Specificity; D013997:Time Factors; D019210:Troponin I / Q000276:immunology / Q000378:metabolism*
D000911:Antibodies, Monoclonal; D019210:Troponin I