title: Advocating asymmetry and the POP-1 paradox: noncanonical Wnt signaling in C. elegans.
abstract: In this issue of Cell, Kidd and colleagues (Kidd et al., 2005) describe their identification of a novel beta-catenin that functions in noncanonical C. elegans Wnt signaling pathways to specify the different fates of daughter cells produced by asymmetric cell division.
authors.affiliation: Institute of Molecular Biology, University of Oregon, Eugene, Oregon 97403, USA.
authors.forename: Bruce
authors.initials: B
authors.lastname: Bowerman
mesh_terms: D000818:Animals; D019521:Body Patterning / Q000502:physiology*; D017173:Caenorhabditis elegans / Q000196:embryology / Q000378:metabolism*; D029742:Caenorhabditis elegans Proteins / Q000378:metabolism*; D003598:Cytoskeletal Proteins / Q000378:metabolism; D004268:DNA-Binding Proteins / Q000378:metabolism*; D006609:High Mobility Group Proteins / Q000378:metabolism*; D036341:Intercellular Signaling Peptides and Proteins / Q000378:metabolism*; D051153:Wnt Proteins
chemical_list: D029742:Caenorhabditis elegans Proteins; D003598:Cytoskeletal Proteins; D004268:DNA-Binding Proteins; D006609:High Mobility Group Proteins; D036341:Intercellular Signaling Peptides and Proteins; C120104:WRM-1 protein, C elegans; D051153:Wnt Proteins; C096787:pop-1 protein, C elegans