Extended probit mortality model for zooplankton against transient change of PCO(2).
The direct injection of CO(2) in the deep ocean is a promising way to mitigate global warming. One of the uncertainties in this method, however, is its impact on marine organisms in the near field. Since the concentration of CO(2), which organisms experience in the ocean, changes with time, it is required to develop a biological impact model for the organisms against the unsteady change of CO(2) concentration. In general, the LC(50) concept is widely applied for testing a toxic agent for the acute mortality. Here, we regard the probit-transformed mortality as a linear function not only of the concentration of CO(2) but also of exposure time. A simple mathematical transform of the function gives a damage-accumulation mortality model for zooplankton. In this article, this model was validated by the mortality test of Metamphiascopsis hirsutus against the transient change of CO(2) concentration.
Sato
Toru
T
Department of Environmental Studies, University of Tokyo, 7-3-1 Hongo, Bunkyo-ku, Tokyo 113-8656, Japan. sato-t@k.u-tokyo.ac.jp
Watanabe
Yuji
Y
Toyota
Koji
K
Ishizaka
Joji
J
D000818:Animals; D002245:Carbon Dioxide / Q000032:analysis / Q000633:toxicity*; D033342:Copepoda / Q000187:drug effects / Q000502:physiology*; D007928:Lethal Dose 50; D008954:Models, Biological; D009026:Mortality; D012623:Seawater / Q000737:chemistry*; D015048:Zooplankton / Q000187:drug effects / Q000502:physiology*
D002245:Carbon Dioxide