title: [Study of CTnI immunohistochemistry on acute myocardial infarction in rabbits].
abstract: OBJECTIVE
  To investigate the signification of CTnI in acute myocardial infarction.
  
  
  METHODS
  The model of myocardial infarction was established by ligating the left ventricle branch. Immunohistochemistry and image analysis were used to detect the change of CTnI in the model, and The sensitivity of immunohistochemistry and HE coloration were also compared.
  
  
  RESULTS
  The acute myocardial infarction tissue showed obvious depletion of CTnI, there was no characterization of myocardial infarction in HE coloration.
  
  
  CONCLUSIONS
  CTnI is sensitive to diagnosis of acute myocardial infarction.
authors.affiliation: Department of Forensic Medicine, Shanghai Medical College, Fudan University, Shanghai, China.
authors.forename: Jian-zhang
authors.initials: JZ
authors.lastname: Jia
authors.forename: Zi-qin
authors.initials: ZQ
authors.lastname: Zhao
authors.forename: Yun-ju
authors.initials: YJ
authors.lastname: Gu
mesh_terms: D000818:Animals; D000911:Antibodies, Monoclonal; D004195:Disease Models, Animal; D005260:Female; D007150:Immunohistochemistry; D008297:Male; D009203:Myocardial Infarction / Q000175:diagnosis* / Q000378:metabolism*; D017202:Myocardial Ischemia / Q000378:metabolism / Q000473:pathology; D009206:Myocardium / Q000378:metabolism* / Q000473:pathology; D011817:Rabbits; D012680:Sensitivity and Specificity; D013997:Time Factors; D019210:Troponin I / Q000276:immunology / Q000378:metabolism*
chemical_list: D000911:Antibodies, Monoclonal; D019210:Troponin I