[Renal intraorgan veins in the normal kidney and in ischemic heart disease].
Using macromicroscopic and morphometric methods, the kidneys obtained from 29 mature and elderly persons which died from mechanical asphyxia (control group) and from 31 patients which died from cardiovascular failure that developed against a background of an ischemic heart disease (IHD), were studied. The changes of architectonics of renal venous blood vessels were found to depend on patient's age and IHD form. Most pronounced changes in venous vascular bed architectonics and volume were detected in an acute form of IHD, while those in chronic form of IHD were less expressed. Acute form of IHD was accompanied by a reduction of relative amount of the venous blood vessels in the renal cortex, empty appearance of capillaries and venules. In the medulla, venulae recti were significantly dilated. In chronic form of IHD, the disturbances of venous compartment of microcirculatory bed develop according the juxtamedullary shunting pattern (in 42% of cases) and excessive venous plethora (44% of cases). In 14% of patients many of the venous vessels in both renal cortex and medulla appeared empty.
Kaplunova
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D000208:Acute Disease; D000368:Aged; D002196:Capillaries / Q000473:pathology; D002908:Chronic Disease; D005260:Female; D006801:Humans; D007668:Kidney / Q000098:blood supply*; D007672:Kidney Cortex / Q000098:blood supply; D008297:Male; D008833:Microcirculation / Q000473:pathology; D008875:Middle Aged; D017202:Myocardial Ischemia / Q000473:pathology*; D012082:Renal Veins / Q000473:pathology*; D014699:Venules / Q000473:pathology