[Morphology of surface and atrophic gastritis in eradication of Helicobacter pylori (HP)].
It is shown with the use of immunohistochemical detection of Bcl-2, caspase-3 and Ki-67 that eradication of HP does not result in immediate reverse development of atrophic changes. This depends on duration of persistence of mononuclear inflammatory cells.
Kononov
A V
AV
Mozgovoĭ
S I
SI
Livzan
M A
MA
Predvechnaia
I K
IK
Novikova
L D
LD
D017209:Apoptosis; D053148:Caspase 3; D020169:Caspases / Q000032:analysis*; D002908:Chronic Disease; D005757:Gastritis, Atrophic / Q000209:etiology / Q000276:immunology*; D016481:Helicobacter Infections / Q000150:complications / Q000628:therapy*; D016480:Helicobacter pylori; D006801:Humans; D019394:Ki-67 Antigen / Q000032:analysis*; D009000:Monocytes / Q000276:immunology; D019253:Proto-Oncogene Proteins c-bcl-2 / Q000032:analysis*; D012038:Regeneration; D013270:Stomach / Q000276:immunology / Q000473:pathology / Q000502:physiology*
D019394:Ki-67 Antigen; D019253:Proto-Oncogene Proteins c-bcl-2; C505407:CASP3 protein, human; D053148:Caspase 3; D020169:Caspases