Lamellar interface fluid accumulation following traumatic corneal perforation and laser in situ keratomileusis.
A 41-year-old man with myopic astigmatism had laser in situ keratomileusis (LASIK) in each eye in April 2002. Ten months later, he sustained a central perforating corneal injury to the right eye. One day following repair of the corneal wound, he presented with diffuse corneal epithelial microcystic edema, lamellar interface fluid accumulation, and 20/400 visual acuity. Additional sutures were placed to close a presumed posterior wound gape with complete resolution of the corneal edema and lamellar interface fluid collection. One year later, his best corrected visual acuity measured 20/20+ in the right eye. This case is the first to document lamellar interface fluid accumulation following LASIK owing to traumatic disruption of the corneal endothelium.
Bushley
D Matthew
DM
Ophthalmology Service, Tripler Army Medical Center , Honolulu, Hawaii 96859, USA.
Holzinger
Karl A
KA
Winkle
R Kevin
RK
Le
Lam H
LH
Olkowski
John D
JD
D000328:Adult; D001251:Astigmatism / Q000601:surgery; D001826:Body Fluids / Q000378:metabolism*; D003315:Cornea / Q000378:metabolism; D015715:Corneal Edema / Q000209:etiology* / Q000378:metabolism / Q000601:surgery; D065306:Corneal Injuries; D057112:Corneal Perforation / Q000209:etiology* / Q000601:surgery; D004728:Endothelium, Corneal / Q000378:metabolism / Q000473:pathology*; D006801:Humans; D020731:Keratomileusis, Laser In Situ; D008297:Male; D009216:Myopia / Q000601:surgery; D013524:Surgical Flaps; D014792:Visual Acuity / Q000502:physiology