title: Clinical examples of disturbed IGF signaling: intrauterine and postnatal growth retardation due to mutations of the insulin-like growth factor I receptor (IGF-IR) gene.
authors.affiliation: Hospital for Children and Adolescents, University of Leipzig, Oststr. 21-25, D-04317, Leipzig, Germany. kiw@medizin.uni-leipzig.de
authors.forename: W
authors.initials: W
authors.lastname: Kiess
authors.forename: J
authors.initials: J
authors.lastname: Kratzsch
authors.forename: E
authors.initials: E
authors.lastname: Keller
authors.forename: A
authors.initials: A
authors.lastname: Schneider
authors.forename: K
authors.initials: K
authors.lastname: Raile
authors.forename: J
authors.initials: J
authors.lastname: Klammt
authors.forename: B
authors.initials: B
authors.lastname: Seidel
authors.forename: A
authors.initials: A
authors.lastname: Garten
authors.forename: H
authors.initials: H
authors.lastname: Schmidt
authors.forename: R
authors.initials: R
authors.lastname: Pfäffle
mesh_terms: D002884:Chromosomes, Human, Pair 15 / Q000235:genetics; D005317:Fetal Growth Retardation / Q000235:genetics*; D006130:Growth Disorders / Q000235:genetics*; D006579:Heterozygote; D006801:Humans; D007223:Infant; D007334:Insulin-Like Growth Factor I / Q000502:physiology*; D008297:Male; D010641:Phenotype; D017354:Point Mutation; D017526:Receptor, IGF Type 1 / Q000096:biosynthesis / Q000235:genetics*
chemical_list: D007334:Insulin-Like Growth Factor I; D017526:Receptor, IGF Type 1