Hepatocavopathy and isolated splenic vein thrombosis due to hypercoagulability state.
We report a patient with protein C and protein S deficiency and factor V Leiden mutation presenting with splenic vein thrombosis and with a web between the hepatic venous confluence and vena cava inferior. These two findings were thought to be due to the hypercoagulable state of the patient. Interestingly, there was no need for invasive procedures as the inferior accessory hepatic vein was patent. Hepatic venous flow was being maintained by the inferior accessory hepatic vein or a dominant collateral vein.
Akarsu
Mesut
M
Department of Gastroenterology, Faculty of Medicine, Dokuz Eylül University, Izmir, Turkey. mesut.akarsu@deu.edu.tr
Seçil
Mustafa
M
Yener
Serkan
S
Topalak
Omer
O
D000925:Anticoagulants / Q000627:therapeutic use; D005260:Female; D006801:Humans; D008102:Liver Circulation; D008875:Middle Aged; D013162:Splenic Vein / Q000473:pathology* / Q000503:physiopathology; D019851:Thrombophilia / Q000150:complications*; D014682:Vena Cava, Inferior / Q000503:physiopathology; D020246:Venous Thrombosis / Q000175:diagnosis* / Q000188:drug therapy / Q000209:etiology / Q000473:pathology / Q000503:physiopathology; D014859:Warfarin / Q000627:therapeutic use
D000925:Anticoagulants; D014859:Warfarin