title: The electrophysiology of atrioventricular nodal reentry tachycardia following the Mustard or Senning procedure and its radiofrequency ablation.
abstract: We describe the electrophysiological studies undertaken in four patients with atrioventricular nodal reentry tachycardia in the setting of concordant atrioventricular and discordant ventriculo-arterial connections (transposition). Radiofrequency ablation was attempted in three, all with success. Clear evidence of dual antegrade pathways through the atrioventricular node was present in only one of the four, but other characteristics of discrete fast and slow pathways into the atrioventricular node were present in all. Atrioventricular nodal reentry tachycardia was inducible in all. In the three patients in whom ablation was attempted, the application of radiofrequency energy to the low medial regions of the systemic venous atrium (morphologically left) consistently caused junctional accelerated rhythm, but these lesions were not successful in eliminating the tachycardia. Successful radiofrequency ablation required a retrograde approach to the region of the slow pathway in the pulmonary venous atrium (morphologically right).
authors.affiliation: Department of Pediatrics, Division of Cardiology, Stanford University, Stanford, California, USA. anne.greene@med.nyu.edu
authors.forename: Anne E
authors.initials: AE
authors.lastname: Greene
authors.forename: Jonathan R
authors.initials: JR
authors.lastname: Skinner
authors.forename: Anne M
authors.initials: AM
authors.lastname: Dubin
authors.forename: Kathryn K
authors.initials: KK
authors.lastname: Collins
authors.forename: George F
authors.initials: GF
authors.lastname: Van Hare
mesh_terms: D000293:Adolescent; D000328:Adult; D002304:Cardiac Pacing, Artificial; D017115:Catheter Ablation; D004562:Electrocardiography; D005471:Fluoroscopy; D006801:Humans; D007223:Infant; D013611:Tachycardia, Atrioventricular Nodal Reentry / Q000150:complications / Q000503:physiopathology / Q000601:surgery*; D014188:Transposition of Great Vessels / Q000150:complications / Q000503:physiopathology; D016896:Treatment Outcome