The proinflammatory heart failure phenotype: a case of integrative physiology.
Weber
Karl T
KT
Division of Cardiovascular Diseases, University of Tennessee Health Science Center, Memphis, Tennessee 38163, USA. KTWeber@utmem.edu
D000450:Aldosterone / Q000097:blood; D000818:Animals; D002118:Calcium / Q000502:physiology; D003326:Coronary Circulation / Q000502:physiology*; D016207:Cytokines / Q000276:immunology; D004195:Disease Models, Animal; D006333:Heart Failure / Q000097:blood / Q000209:etiology* / Q000276:immunology / Q000503:physiopathology; D006801:Humans; D006929:Hyperaldosteronism / Q000097:blood / Q000150:complications / Q000276:immunology; D006961:Hyperparathyroidism / Q000097:blood / Q000150:complications / Q000276:immunology; D007249:Inflammation / Q000150:complications / Q000276:immunology*; D008274:Magnesium / Q000502:physiology; D018384:Oxidative Stress / Q000276:immunology; D010641:Phenotype
D016207:Cytokines; D000450:Aldosterone; D008274:Magnesium; D002118:Calcium