title: IgH/TCR rearrangements are common in MLL translocated adult AML and suggest an early T/myeloid or B/myeloid maturation arrest, which correlates with the MLL partner.
authors.forename: C
authors.initials: C
authors.lastname: Dupret
authors.forename: V
authors.initials: V
authors.lastname: Asnafi
authors.forename: D
authors.initials: D
authors.lastname: Leboeuf
authors.forename: C
authors.initials: C
authors.lastname: Millien
authors.forename: R
authors.initials: R
authors.lastname: Ben Abdelali
authors.forename: C
authors.initials: C
authors.lastname: Preudhomme
authors.forename: K
authors.initials: K
authors.lastname: Beldjord
authors.forename: E
authors.initials: E
authors.lastname: Delabesse
authors.forename: E
authors.initials: E
authors.lastname: Macintyre
mesh_terms: D000208:Acute Disease; D000328:Adult; D001402:B-Lymphocytes / Q000473:pathology; D019070:Cell Lineage; D015321:Gene Rearrangement; D019672:Genes, T-Cell Receptor; D006801:Humans; D007143:Immunoglobulin Heavy Chains / Q000235:genetics; D007951:Leukemia, Myeloid / Q000235:genetics / Q000473:pathology*; D022423:Myeloid Cells / Q000473:pathology; D051788:Myeloid-Lymphoid Leukemia Protein / Q000235:genetics*; D013601:T-Lymphocytes / Q000473:pathology; D014178:Translocation, Genetic
chemical_list: D007143:Immunoglobulin Heavy Chains; D051788:Myeloid-Lymphoid Leukemia Protein